Dr Jessie Sutherland
ARC DECRA
School of Biomedical Sciences and Pharmacy
- Email:jessie.sutherland@newcastle.edu.au
- Phone:(02) 4913 8735
Fertile grounds for research into biological sciences
Ever since her first experiences as an undergraduate Science student at UON, Dr Jessie Sutherland remembers being inspired by reproductive biology.
“The (Reproductive Science) group that we have here is just amazing. It is really inspiring.”
Jessie’s academic career started when she came across a summer scholarship program supervised by Conjoint-Professor Eileen McLaughlin, who has been her mentor ever since.
“I found her really engaging. There were hundreds of students in those first year lectures and she was still very engaging and personable.”
Jessie’s early experiences with UON’s Reproductive Science lecturers have inspired her attitudes and teaching practice today.
“Teaching is one of my favourite things."
“It’s one of the most rewarding and positive experiences that you will have in academia, where you are often dealt harsh criticism in the form of grant rebuttals, peer review and article rejection."
“I’m very passionate about the subject material and I think that really comes across to the students. It’s beautiful to have them engaged and asking questions."
“Teaching is definitely something I want to continue to do in the future.”
Indeed, Jessie is continually improving her teaching and leadership skills through her participation in the Advanced Leadership Program. The mentoring program is designed for women working in the higher education sector and Jessie was able to apply for a place with her HMRI Equal Futures grant which she won this year.
Jessie is also working to develop a new Pregnancy and Development course, focussing on online content which will enable more students to undertake studies, as part of the new program structure of UON’s Biomedical Science degree.
As well as her passion for teaching, Jessie has had great success in her research career, which has focused on the JAK/STAT molecular pathway in ovarian development. She has also spent time investigating the role of Musashi proteins in spermatogenesis, the process by which spermatozoa are developed.
Spermatogenesis is dependent on the maintenance of a population of stem cells, known as spermatogonia. Stem cells are distinctive in that they have the potential to divide and give rise to daughter cells with specific characteristics; in this instance - spermatozoa.
Spermatogonia go through long periods of relative ‘genetic silence’. Whereas many cell types are more or less constantly reading their DNA and producing proteins, there are huge swaths of genetic material in the spermatogonia which are left untouched for long periods of time. This is important for the proper development of the male germ cells. Amongst the many factors which control this silence are the Musashi RNA binding proteins. Throughout Jessie’s PhD, she examined the regulation of these binding proteins, and the exact roles they play in germ cell development.
Jessie also researches female fertility. In particular, she is interested in ovarian development and premature ovarian failure.
Jessie is looking forward to returning to this area of research as part of her recent successful NHMRC Early Career Fellowship application.
“I’m so happy that this project got up – it’s sort of a pet project that I’ve been dipping in and out of since Honours.
“We hadn’t really been able to fully explore it to the extent I’d like to – but last year I was fortunate enough to get a small project grant from the HMRI Bob and Terry Kennedy Children’s Research Program. We were able to generate some pilot data from that and I think that was what really pushed me over the line for the NHMRC funding.
“It’s so fantastic that now I have the opportunity and freedom to explore all the ideas I’ve had for so long.”
During the HMRI funded project, Jessie and her team honed in on the specifics of the JAK/STAT molecular pathway during ovarian development. The newly funded NHMRC project will allow them to study global changes which occur at this time.
Premature ovarian failure can be actually be triggered very early during the development of female reproductive organs.
“We’re looking for key factors that we could potentially manipulate in order to prolong fertility in these individuals.”
Outside of the lab and the lecture theaters of UON, Jessie is also an enthusiastic science communicator, and enjoys engaging with the public and talking about her research. In particular, she is keen to educate women about their fertility, as messages perpetuated by popular culture can often be misleading.
“The biggest factor (in female fertility) is age. The limiting factor is the number of eggs you have in your ovaries – and that is indiscriminate of how healthy you are.
“Health can of course detrimentally affect fertility but even if you have a healthy lifestyle, it doesn’t necessarily mean that you will have prolonged fertility.”
Fertile grounds for research into biological sciences
Jessie has focussed on the JAK/STAT molecular pathway in ovarian development.
Career Summary
Biography
Biography
I am extremely passionate about scientific research with my particular focus in the field of reproductive biology, infertility and disease.
Having both worked and studied within the HMRI Pregnancy and Reproductive Program and the PRC in Reproductive Science since 2009 I have a strong background in reproduction, evolving from my involvement in multiple research projects in the areas of; ovarian and testis biology, reproductive toxicology, sexual health, infertility, reproductive cancer, and developmental biology. While my broader research knowledge extends to; gene regulation, cell biology, cell signalling pathways, molecular characterisation, proteomics, and transgenic animal models. I am particularly focused on facilitating the translation of my basic research program into outcomes that directly impact the health and well-being of the wider community. As an ongoing member of the Australian Society of Reproduction I also am part of a strong network of scientific professionals spanning multiple universities in Australia and internationally.
Currently I am working on a number projects that focus on understanding the underlying factors that lead to development of infertility and diseases of the reproductive system. I am specifically interested in determining the factors responsible for premature ovarian failure and the onset of early menopause in young women.
Research Expertise
I obtained my PhD from the University of Newcastle (2015) for exploring the essential role of the Musashi family of RNA binding proteins in controlling mammalian spermatogenesis and fertility. Prior to undertaking my PhD I worked as a Research Assistant within the PRC for Reproductive Science. My Honours project (2010) focused on exploring the molecular mechanisms surrounding the JAK/STAT/SOCS cytokine signally pathway in ovarian development, particularly during primordial follicle activation.
Teaching Expertise
I lecture into the undergraduate Biomedical Science and Biology Programs and Pharmacy.
I am always looking to attract talented students considering a career path in research to meet/join my team and I have a number of Honours and RHD projects advertised through both the School of Biomedical Sciences & Pharmacy and The School of Environmental & Life Sciences. I'm more than happy to meet up with any interested students to discuss our ongoing projects in detail.
Qualifications
- Doctor of Philosophy, University of Newcastle
- Bachelor of Science, University of Newcastle
- Bachelor of Science (Honours), University of Newcastle
Keywords
- Biology
- Infertility
- Molcular biology
- Ovarian biology
- Reproductive Biology
- Reproductive Science
- Spermatogenesis
Fields of Research
Code | Description | Percentage |
---|---|---|
310903 | Animal developmental and reproductive biology | 80 |
310902 | Animal cell and molecular biology | 20 |
Professional Experience
Academic appointment
Dates | Title | Organisation / Department |
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1/12/2014 - 28/2/2015 | Research Academic (full-time) | Priority Research Centre in Reproductive Science Australia |
1/9/2012 - 30/11/2014 | Research Assistant (casual) | Priority Research Centre in Reproductive Science Australia |
1/11/2010 - 31/8/2012 | Research Assistant (full-time) | Priority Research Centre in Reproductive Science Australia |
Professional appointment
Dates | Title | Organisation / Department |
---|---|---|
1/9/2009 - 28/2/2010 | Laboratory Technician | Priority Research Centre in Reproductive Science Australia |
Awards
Award
Year | Award |
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2014 |
Society for Reproductive Biology Early Career Researcher Travel Award Society for Reproductive Biology |
2014 |
Society for Reproductive Biology New Investigator Award Society for Reproductive Biology |
2010 |
UON Faculty of Science & IT Medal The University of Newcastle |
Distinction
Year | Award |
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2013 |
Society for Reproductive Biology Ozooa Student Award - Finalist Society for Reproductive Biology |
Research Award
Year | Award |
---|---|
2014 |
Faculty Award for Outstanding Postgraduate (Research) Student Achievement The University of Newcastle |
Scholarship
Year | Award |
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2014 |
UON Faculty of Science & IT RHD Conference Scholarship The University of Newcastle |
2012 |
UON School of Environmental and Life Sciences RHD funding The University of Newcastle |
2010 |
UON Faculty of Science & IT Honours Scholarship The University of Newcastle |
2008 |
UON Faculty of Science & IT Summer Vacation Scholarship The University of Newcastle |
Publications
For publications that are currently unpublished or in-press, details are shown in italics.
Chapter (1 outputs)
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2013 |
Sutherland JM, McLaughlin EA, Hime GR, Siddall NA, 'The Musashi family of RNA binding proteins: Master regulators of multiple stem cell populations', 233-245 (2013) [B1] In order to maintain their unlimited capacity to divide, stem cells require controlled temporal and spatial protein expression. The Musashi family of RNA-binding proteins have bee... [more] In order to maintain their unlimited capacity to divide, stem cells require controlled temporal and spatial protein expression. The Musashi family of RNA-binding proteins have been shown to exhibit this necessary translational control through both repression and activation in order to regulate multiple stem cell populations. This chapter looks in depth at the initial discovery and characterisation of Musashi in the model organism Drosophila, and its subsequent emergence as a master regulator in a number of stem cell populations. Furthermore the unique roles for mammalian Musashi-1 and Musashi-2 in different stem cell types are correlated with the perceived diagnostic power of Musashi expression in specific stem cell derived oncologies. In particular the potential role for Musashi in the identification and treatment of human cancer is considered, with a focus on the role of Musashi-2 in leukaemia. Finally, the manipulation of Musashi expression is proposed as a potential avenue towards the targeted treatment of specific aggressive stem cell cancers. © 2013 Springer Science+Business Media Dordrecht.
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Journal article (42 outputs)
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2023 |
Frost ER, Ford EA, Peters AE, Lovell-Badge R, Taylor G, McLaughlin EA, Sutherland JM, 'A New Understanding, Guided by Single-Cell Sequencing, of the Establishment and Maintenance of the Ovarian Reserve in Mammals', SEXUAL DEVELOPMENT, 17 145-155 (2023) [C1]
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2023 |
Radhakrishnan K, Luu M, Iaria J, Sutherland JM, McLaughlin EA, Zhu HJ, Loveland KL, 'Activin and BMP Signalling in Human Testicular Cancer Cell Lines, and a Role for the Nucleocytoplasmic Transport Protein Importin-5 in Their Crosstalk', Cells, 12 (2023) [C1] Testicular germ cell tumours (TGCTs) are the most common malignancy in young men. Originating from foetal testicular germ cells that fail to differentiate correctly, TGCTs appear ... [more] Testicular germ cell tumours (TGCTs) are the most common malignancy in young men. Originating from foetal testicular germ cells that fail to differentiate correctly, TGCTs appear after puberty as germ cell neoplasia in situ cells that transform through unknown mechanisms into distinct seminoma and non-seminoma tumour types. A balance between activin and BMP signalling may influence TGCT emergence and progression, and we investigated this using human cell line models of seminoma (TCam-2) and non-seminoma (NT2/D1). Activin A- and BMP4-regulated transcripts measured at 6 h post-treatment by RNA-sequencing revealed fewer altered transcripts in TCam-2 cells but a greater responsiveness to activin A, while BMP4 altered more transcripts in NT2/D1 cells. Activin significantly elevated transcripts linked to pluripotency, cancer, TGF-ß, Notch, p53, and Hippo signalling in both lines, whereas BMP4 altered TGF-ß, pluripotency, Hippo and Wnt signalling components. Dose-dependent antagonism of BMP4 signalling by activin A in TCam-2 cells demonstrated signalling crosstalk between these two TGF-ß superfamily arms. Levels of the nuclear transport protein, IPO5, implicated in BMP4 and WNT signalling, are highly regulated in the foetal mouse germline. IPO5 knockdown in TCam-2 cells using siRNA blunted BMP4-induced transcript changes, indicating that IPO5 levels could determine TGF-ß signalling pathway outcomes in TGCTs.
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2023 |
Ford EA, Chojenta C, Bagade T, Sweeney S, Sutherland JM, 'Fertility knowledge in a cohort of Australia s adolescents: a cross-sectional study of reproductive and sexual health education', Sex Education, 1-19 [C1]
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2022 |
Ford EA, Frost ER, Beckett EL, Roman SD, McLaughlin EA, Sutherland JM, 'Transcriptomic profiling of neonatal mouse granulosa cells reveals new insights into primordial follicle activation .', Biol Reprod, 106 503-514 (2022) [C1]
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2022 |
Siddall NA, Casagranda F, Johanson TM, Dominado N, Heaney J, Sutherland JM, et al., 'MiMIC analysis reveals an isoform specific role for Drosophila Musashi in follicle stem cell maintenance and escort cell function', CELL DEATH DISCOVERY, 8 (2022) [C1]
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2022 |
Ford EA, Peters AE, Roman SD, McLaughlin EA, Beckett EL, Sutherland JM, 'A scoping review of the information provided by fertility smartphone applications', Human Fertility, 25 625-639 (2022) [C1] The growth of smartphone application use across areas of female reproductive health has led to increased interest into their functions and benefits. This scoping review aims to de... [more] The growth of smartphone application use across areas of female reproductive health has led to increased interest into their functions and benefits. This scoping review aims to determine the nature and extent of the peer-reviewed literature presented on fertility-based apps, to identify the reliability of the information within the apps, and to determine the ability of this information to educate users. A systematic search of six databases was conducted in April 2020, returning a total of 21,158 records. After duplicate removal, title and abstract screening exclusionary steps, 27 records were reviewed and charted. Records covered a variety of reproductive health themes including contraception, sexual health, and family planning, and used a range of methodologies. The accuracy of fertility information within the apps reported in these studies was variable, but overall there was a lack of depth in the coverage of content in apps. It was common for studies in this review to base fertile window algorithms on stringent cycle length and variability requirements, limiting the applicability of information delivered to users. Furthermore, studies from app affiliates often lacked collaborations with researchers, minimising the potential for fertility knowledge improvements integrated across the suite of female reproductive health apps.
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2021 |
Sutherland JM, McLaughlin EA, 'Ovarian ageing: Where are we now? And where to next?', Current Opinion in Endocrine and Metabolic Research, 18 29-34 (2021) [C1] From the mid-1980s, the scientific community has demonstrated irrefutable evidence for the age-related decline in female fertility (Menken et al., 1986) [1], supporting observatio... [more] From the mid-1980s, the scientific community has demonstrated irrefutable evidence for the age-related decline in female fertility (Menken et al., 1986) [1], supporting observations in animals and humans of maternal ageing, increased incidence of miscarriage and congenital abnormality dating back to the mid-20th Century (Warburton, 1970) [2]. Although, it was not until 2005 that a report arising from the European Society of Human Reproduction and Embryology sought to address ovarian ageing as the primary contributing factor to this decline and its profound influence on the success of assisted reproduction. Over the resulting two decades, researchers have endeavoured to define and quantify the genetic, molecular and cellular factors directly responsible for ovarian ageing and subsequently female age-related infertility, seeking to ultimately improve diagnostics and treatments. In this review, we undertake a closer investigation of the ¿usual suspects¿ and speculate on the role of synergistic inflammaging and finally address the question of whether in 2020 we are really any closer to finding the culprits underpinning the ageing ovary.
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2021 |
Walters JLH, Anderson AL, da Silva SJM, Aitken RJ, De Iuliis GN, Sutherland JM, et al., 'Mechanistic insight into the regulation of lipoxygenase-driven lipid peroxidation events in human spermatozoa and their impact on male fertility', Antioxidants, 10 1-19 (2021) [C1] A prevalent cause of sperm dysfunction in male infertility patients is the overproduction of reactive oxygen species, an attendant increase in lipid peroxidation and the productio... [more] A prevalent cause of sperm dysfunction in male infertility patients is the overproduction of reactive oxygen species, an attendant increase in lipid peroxidation and the production of cyto-toxic reactive carbonyl species such as 4-hydroxynonenal. Our previous studies have implicated arachidonate 15-lipoxygenase (ALOX15) in the production of 4-hydroxynonenal in developing germ cells. Here, we have aimed to develop a further mechanistic understanding of the lipoxygen-ase-lipid peroxidation pathway in human spermatozoa. Through pharmacological inhibition studies, we identified a protective role for phospholipase enzymes in the liberation of peroxidised polyunsaturated fatty acids from the human sperm membrane. Our results also revealed that arachi-donic acid, linoleic acid and docosahexanoic acid are key polyunsaturated fatty acid substrates for ALOX15. Upon examination of ALOX15 in the spermatozoa of infertile patients compared to their normozoospermic counterparts, we observed significantly elevated levels of ALOX15 protein abundance in the infertile population and an increase in 4-hydroxynonenal adducts. Collectively, these data confirm the involvement of ALOX15 in the oxidative stress cascade of human spermatozoa and support the notion that increased ALOX15 abundance in sperm cells may accentuate membrane lipid peroxidation and cellular dysfunction, ultimately contributing to male infertility.
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2021 |
Peters AE, Caban SJ, McLaughlin EA, Roman SD, Bromfield EG, Nixon B, Sutherland JM, 'The Impact of Aging on Macroautophagy in the Pre-ovulatory Mouse Oocyte', Frontiers in Cell and Developmental Biology, 9 (2021) [C1] Accompanying the precipitous age-related decline in human female fertility is an increase in the proportion of poor-quality oocytes within the ovary. The macroautophagy pathway, a... [more] Accompanying the precipitous age-related decline in human female fertility is an increase in the proportion of poor-quality oocytes within the ovary. The macroautophagy pathway, an essential protein degradation mechanism responsible for maintaining cell health, has not yet been thoroughly investigated in this phenomenon. The aim of this study was to characterize the macroautophagy pathway in an established mouse model of oocyte aging using in-depth image analysis-based methods and to determine mechanisms that account for the observed changes. Three autophagy pathway markers were selected for assessment of gene and protein expression in this model: Beclin 1; an initiator of autophagosome formation, Microtubule-associated protein 1 light chain 3B; a constituent of the autophagosome membrane, and lysosomal-associated membrane protein 1; a constituent of the lysosome membrane. Through quantitative image analysis of immunolabeled oocytes, this study revealed impairment of the macroautophagy pathway in the aged oocyte with an attenuation of both autophagosome and lysosome number. Additionally, an accumulation of amphisomes greater than 10 µm2 in area were observed in aging oocytes, and this accumulation was mimicked in oocytes treated with lysosomal inhibitor chloroquine. Overall, these findings implicate lysosomal dysfunction as a prominent mechanism by which these age-related changes may occur and highlight the importance of macroautophagy in maintaining mouse pre-ovulatory oocyte quality. This provides a basis for further investigation of dysfunctional autophagy in poor oocyte quality and for the development of therapeutic or preventative strategies to aid in the maintenance of pre-ovulatory oocyte health.
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2021 |
Fraser B, Peters AE, Sutherland JM, Liang M, Rebourcet D, Nixon B, Aitken RJ, 'Biocompatible Nanomaterials as an Emerging Technology in Reproductive Health; a Focus on the Male', Frontiers in Physiology, 12 (2021) [C1] A growing body of research has confirmed that nanoparticle (NP) systems can enhance delivery of therapeutic and imaging agents as well as prevent potentially damaging systemic exp... [more] A growing body of research has confirmed that nanoparticle (NP) systems can enhance delivery of therapeutic and imaging agents as well as prevent potentially damaging systemic exposure to these agents by modifying the kinetics of their release. With a wide choice of NP materials possessing different properties and surface modification options with unique targeting agents, bespoke nanosystems have been developed for applications varying from cancer therapeutics and genetic modification to cell imaging. Although there remain many challenges for the clinical application of nanoparticles, including toxicity within the reproductive system, some of these may be overcome with the recent development of biodegradable nanoparticles that offer increased biocompatibility. In recognition of this potential, this review seeks to present recent NP research with a focus on the exciting possibilities posed by the application of biocompatible nanomaterials within the fields of male reproductive medicine, health, and research.
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2021 |
Frost ER, Taylor G, Baker MA, Lovell-Badge R, Sutherland JM, 'Establishing and maintaining fertility: the importance of cell cycle arrest', GENES & DEVELOPMENT, 35 619-634 (2021) [C1]
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2021 |
Frost ER, Ford EA, Taylor G, Boeing S, Beckett EL, Roman SD, et al., 'Two alternative methods for the retrieval of somatic cell populations from the mouse ovary', MOLECULAR HUMAN REPRODUCTION, 27 (2021) [C1]
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2020 |
Ford E, Beckett EL, Roman S, McLaughlin EA, Sutherland J, 'Advances in human primordial follicle activation and premature ovarian insufficiency.', Reproduction, 159 R15-R29 (2020) [C1]
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2020 |
Walters JLH, Gadella BM, Sutherland JM, Nixon B, Bromfield EG, 'Male Infertility: Shining a Light on Lipids and Lipid-Modulating Enzymes in the Male Germline', Journal of Clinical Medicine, 9 (2020) [C1]
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2020 |
Peters AE, Mihalas BP, Bromfield EG, Roman SD, Nixon B, Sutherland JM, 'Autophagy in Female Fertility: A Role in Oxidative Stress and Aging', ANTIOXIDANTS & REDOX SIGNALING, 32 550-568 (2020) [C1]
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2020 |
Frost ER, Ford EA, Peters AE, Reed NL, McLaughlin EA, Baker MA, et al., 'Signal transducer and activator of transcription (STAT) 1 and STAT3 are expressed in the human ovary and have Janus kinase 1-independent functions in the COV434 human granulosa cell line', Reproduction, Fertility and Development, 32 1027-1039 (2020) [C1]
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2020 |
Ford EA, Roman SD, McLaughlin EA, Beckett EL, Sutherland JM, 'The association between reproductive health smartphone applications and fertility knowledge of Australian women', BMC Women's Health, 20 (2020) [C1]
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2020 |
Bryan ER, Redgrove KA, Mooney AR, Mihalas BP, Sutherland JM, Carey AJ, et al., 'Chronic testicular Chlamydia muridarum infection impairs mouse fertility and offspring development ', Biology of Reproduction, 102 888-901 (2020) [C1]
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2020 |
Mihalas BP, Redgrove KA, Bernstein IR, Robertson MJ, McCluskey A, Nixon B, et al., 'Dynamin 2-dependent endocytosis is essential for mouse oocyte development and fertility', FASEB Journal, 34 5162-5177 (2020) [C1]
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2019 |
Martin JH, Aitken RJ, Bromfield E, Cafe SL, Sutherland JM, Frost ER, et al., 'Investigation into the presence and functional significance of proinsulin C-peptide in the female germline', Biology of Reproduction, 100 1275-1289 (2019) [C1]
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2019 |
Mihalas BP, Camlin NJ, Xavier MJ, Peters AE, Holt JE, Sutherland JM, et al., 'The small non-coding RNA profile of mouse oocytes is modified during aging', AGING-US, 11 2968-2997 (2019) [C1]
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2018 |
Sutherland JM, Sobinoff AP, Fraser BA, Redgrove KA, Siddall NA, Koopman P, et al., 'RNA binding protein Musashi-2 regulates PIWIL1 and TBX1 in mouse spermatogenesis', JOURNAL OF CELLULAR PHYSIOLOGY, 233 3262-3273 (2018) [C1]
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2018 |
Saeidi S, Shapouri F, de Iongh RU, Casagranda F, Sutherland JM, Western PS, et al., 'Esrp1 is a marker of mouse fetal germ cells and differentially expressed during spermatogenesis', PLOS ONE, 13 (2018) [C1]
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2018 |
Hall SE, Upton RMO, McLaughlin EA, Sutherland JM, 'Phosphoinositide 3-kinase/protein kinase B (PI3K/AKT) and Janus kinase/signal transducer and activator of transcription (JAK/STAT) follicular signalling is conserved in the mare ovary', Reproduction, Fertility and Development, 30 624-633 (2018) [C1] The mare ovary is unique in its anatomical structure; however, the signalling pathways responsible for physiological processes, such as follicular activation, remain uncharacteris... [more] The mare ovary is unique in its anatomical structure; however, the signalling pathways responsible for physiological processes, such as follicular activation, remain uncharacterised. This provided us with the impetus to explore whether signalling molecules from important folliculogenesis pathways, phosphoinositide 3-kinase/protein kinase B (PI3K/AKT) and Janus kinase/signal transducer and activator of transcription (JAK/STAT), are conserved in the mare ovary. Messenger RNA expression of six genes important in follicle development was measured using quantitative polymerase chain reaction and protein localisation of key pathway members (PI3K, AKT1, phosphatase and tensin homologue (PTEN), JAK1, STAT3 and suppressor of cytokine signalling 4 (SOCS4)) was compared in tissue from fetal and adult mare ovaries. Tissue from adult ovaries exhibited significantly increased levels of mRNA expression of PI3K, AKT1, PTEN, JAK1, STAT3 and SOCS4 compared with tissue from fetal ovaries. PI3K, AKT1, JAK1 and STAT3 demonstrated redistributed localisation, from pregranulosa cells in fetal development, to both the oocyte and granulosa cells of follicles in the adult ovary, whilst negative feedback molecules PTEN and SOCS4 were only localised to the granulosa cells in the adult ovary. These findings suggest that the PI3K/AKT and JAK/STAT signalling pathways are utilised during folliculogenesis in the mare, similarly to previously studied mammalian species, and may serve as useful biomarkers for assessment of ovary development in the horse.
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2018 |
Lucock M, Thota R, Garg M, Martin C, Jones P, Furst J, et al., 'Vitamin D and folate: A reciprocal environmental association based on seasonality and genetic disposition', AMERICAN JOURNAL OF HUMAN BIOLOGY, 30 (2018) [C1]
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2018 |
Sutherland JM, Frost ER, Ford EA, Peters AE, Reed NL, Seldon AN, et al., 'Janus Kinase JAK1 maintains the ovarian reserve of primordial follicles in the mouse ovary.', Molecular Human Reproduction, 24 533-542 (2018) [C1]
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2018 |
Mihalas BP, Bromfield EG, Sutherland JM, De Iuliis GN, McLaughlin EA, John Aitken R, Nixon B, 'Oxidative damage in naturally aged mouse oocytes is exacerbated by dysregulation of proteasomal activity', Journal of Biological Chemistry, 293 18944-18964 (2018) [C1]
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2017 |
Fuller EA, Sominsky L, Sutherland JM, Redgrove KA, Harms L, McLaughlin EA, Hodgson DM, 'Neonatal immune activation depletes the ovarian follicle reserve and alters ovarian acute inflammatory mediators in neonatal rats', BIOLOGY OF REPRODUCTION, 97 719-730 (2017) [C1]
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2016 |
Redgrove KA, Bernstein IR, Pye VJ, Mihalas BP, Sutherland JM, Nixon B, et al., 'Dynamin 2 is essential for mammalian spermatogenesis', SCIENTIFIC REPORTS, 6 (2016) [C1]
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2016 |
Shapouri F, Saeidi S, de Iongh RU, Casagranda F, Western PS, McLaughlin EA, et al., 'Tob1 is expressed in developing and adult gonads and is associated with the P-body marker, Dcp2', Cell and Tissue Research, 364 443-451 (2016) [C1] Tob1 is a member of the BTG/TOB family of proteins with established antiproliferative function. In Danio rerio and Xenopus laevis, the Tob1 gene is expressed from the one-cell sta... [more] Tob1 is a member of the BTG/TOB family of proteins with established antiproliferative function. In Danio rerio and Xenopus laevis, the Tob1 gene is expressed from the one-cell stage through to early gastrula stages, followed in later development by discrete expression in many tissues including the notochord and somites. In both mouse and human, Tob1 is expressed in many adult tissues including the testis and ovary; however, the specific cell types are unknown. We examine Tob1 gene expression in mouse in developing germ cells and in sorted male germ cells (gonocytes, spermatogonia, pachytene spermatocytes and round spermatids) by reverse transcription and droplet digital polymerase chain reaction (RT-ddPCR) and in adult ovary and testis by immunofluorescence with anti-Tob1 protein staining. By RT-ddPCR, Tob1 expression was low in developing male germ cells but was highly expressed in round spermatids. In developing female germ cells undergoing entry into meiosis, it increased 10-fold. Tob1 was also highly expressed in round spermatids and in oocytes in all stages of folliculogenesis. Notably, a marker for P-bodies, Dcp-2, was also highly expressed in round spermatids and all oocyte stages examined. The cytoplasmic presence of Tob1 protein in round spermatids and oocytes and the association of Tob1 protein with Dcp2 in both cell types suggest that Tob1 protein plays a role in post-transcriptional mechanisms.
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2016 |
Camlin NJ, Sobinoff AP, Sutherland JM, Beckett EL, Jarnicki AG, Vanders RL, et al., 'Maternal Smoke Exposure Impairs the Long-Term Fertility of Female Offspring in a Murine Model.', Biol Reprod, 94 39 (2016) [C1]
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2015 |
Sutherland JM, Sobinoff AP, Fraser BA, Redgrove KA, Davidson TL, Siddall NA, et al., 'RNA binding protein Musashi-1 directly targets Msi2 and Erh during early testis germ cell development and interacts with IPO5 upon translocation to the nucleus', FASEB Journal, 29 2759-2768 (2015) [C1] Controlled gene regulation during gamete development is vital for maintaining reproductive potential. During the process of gamete development, male germ cells experience extended... [more] Controlled gene regulation during gamete development is vital for maintaining reproductive potential. During the process of gamete development, male germ cells experience extended periods of inactive transcription despite requirements for continued growth and differentiation. Spermatogenesis therefore provides an ideal model to study the effects of posttranscriptional control on gene regulation. During spermatogenesis posttranscriptional regulation is orchestrated by abundantly expressed RNA-binding proteins. One such group of RNA-binding proteins is the Musashi family, previously identified as a critical regulator of testis germ cell development and meiosis in Drosophila and also shown to be vital to sperm development and reproductive potential in the mouse. We focus in depth on the role and function of the vertebrate Musashi ortholog Musashi-1 (MSI1). Through detailed expression studies and utilizing our novel transgenic Msi1 testis-specific overexpression model, we have identified 2 unique RNA-binding targets of MSI1 in spermatogonia,Msi2 and Erh, and have demonstrated a role for MSI1 in translational regulation. We have also provided evidence to suggest that nuclear import protein, IPO5, facilitates the nuclear translocation of MSI1 to the transcriptionally silenced XY chromatin domain in meiotic pachytene spermatocytes, resulting in the release of MSI1 RNA-binding targets. This firmly establishes MSI1 as a master regulator of posttranscriptional control during early spermatogenesis and highlights the significance of the subcellular localization of RNA binding proteins in relation to their function.
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2015 |
Sobinoff AP, Dando SJ, Redgrove KA, Sutherland JM, Stanger SJ, Armitage CW, et al., 'Chlamydia muridarum infection-induced destruction of male germ cells and sertoli cells is partially prevented by Chlamydia major outer membrane protein-specific immune CD4 cells.', Biol Reprod, 92 27 (2015) [C1]
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Nova | |||||||||
2015 |
Sutherland JM, Sobinoff AP, Gunter KM, Fraser BA, Pye V, Bernstein IR, et al., 'Knockout of RNA Binding Protein MSI2 Impairs Follicle Development in the Mouse Ovary: Characterization of MSI1 and MSI2 during Folliculogenesis.', Biomolecules, 5 1228-1244 (2015) [C1]
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Nova | |||||||||
2014 |
Sutherland JM, Fraser BA, Sobinoff AP, Pye VJ, Davidson T-L, Siddall NA, et al., 'Developmental expression of Musashi-1 and Musashi-2 RNA-binding proteins during spermatogenesis: analysis of the deleterious effects of dysregulated expression.', Biol Reprod, 90 92 (2014) [C1]
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Nova | |||||||||
2014 |
Sobinoff AP, Sutherland JM, Beckett EL, Stanger SJ, Johnson R, Jarnicki AG, et al., 'Damaging legacy: maternal cigarette smoking has long-term consequences for male offspring fertility.', Hum Reprod, 29 2719-2735 (2014) [C1]
|
Nova | |||||||||
2013 |
Sobinoff AP, Beckett EL, Jarnicki AG, Sutherland JM, McCluskey A, Hansbro PM, McLaughlin EA, 'Scrambled and fried: Cigarette smoke exposure causes antral follicle destruction and oocyte dysfunction through oxidative stress', TOXICOLOGY AND APPLIED PHARMACOLOGY, 271 156-167 (2013) [C1]
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2013 |
Sobinoff AP, Sutherland JM, Mclaughlin EA, 'Intracellular signalling during female gametogenesis', Molecular Human Reproduction, 19 265-278 (2013) [C1] Female reproductive potential is dictated by the size of the primordial follicle pool and the correct regulation of oocyte maturation and activation-events essential for productio... [more] Female reproductive potential is dictated by the size of the primordial follicle pool and the correct regulation of oocyte maturation and activation-events essential for production of viable offspring. Although a substantial body of work underpins our understanding of these processes, the molecular mechanisms of follicular and oocyte development are not fully understood. This review summarizes recent findings which have improved our conception of how folliculogenesis and oocyte competence are regulated, and discusses their implications for assisted reproductive techniques. We highlight evidence provided by genetically modified mouse models and in vitro studies which have refined our understanding of Pi3k/Akt and mTOR signalling in the oocyte and have discovered a role for Jak/Stat/Socs signalling in granulosa cells during primordial follicle activation. We also appraise a novel role for the metal ion zinc in the regulation of meiosis I and meiosis II progression through early meiosis inhibitor (Emi2) and Mos-Mapk signalling, and examine studies which expand our understanding of intracellular calcium signalling and extrinsic Plc¿ in stimulating oocyte activation. © The Author 2012. Published by Oxford University Press on behalf of the European Society of Human Reproduction and Embryology. All rights reserved.
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Nova | |||||||||
2013 |
O'Bryan MK, Clark BJ, McLaughlin EA, D'Sylva RJ, O'Donnell L, Wilce JA, et al., 'RBM5 Is a Male Germ Cell Splicing Factor and Is Required for Spermatid Differentiation and Male Fertility', PLOS GENETICS, 9 (2013) [C1]
|
Nova | |||||||||
2012 |
Sutherland JM, Keightley RA, Nixon B, Roman SD, Robker RL, Russell DL, McLaughlin EA, 'Suppressor of cytokine signaling 4 (SOCS4): Moderator of ovarian primordial follicle activation', Journal of Cellular Physiology, 227 1188-1198 (2012) [C1]
|
Nova | |||||||||
Show 39 more journal articles |
Review (1 outputs)
Year | Citation | Altmetrics | Link | |||||
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2015 |
Sutherland JM, Siddall NA, Hime GR, McLaughlin EA, 'RNA binding proteins in spermatogenesis: an in depth focus on the Musashi family', ASIAN JOURNAL OF ANDROLOGY (2015) [C1]
|
Nova |
Conference (5 outputs)
Year | Citation | Altmetrics | Link | ||
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2017 |
Siddall N, Sutherland J, McLaughlin E, Hime G, 'Drosophila Musashi is required in the ovary to regulate follicle stem cell behaviour and maintain niche homeostasis', MECHANISMS OF DEVELOPMENT, Natil Univ Singapore, Singapore, SINGAPORE (2017)
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2016 | Saeidi SS, Shapouri F, De Iongh R, Casagranda F, Western P, McLaughlin E, et al., 'The role of ESRP1 during gametogenesis', CLINICAL ENDOCRINOLOGY, Adelaide, AUSTRALIA (2016) | ||||
2016 | Shapouri F, Saeidi SS, De Iongh R, Casagranda F, Western PS, McLaughlin E, et al., 'Tob1 protein is a novel regulator of gonadal function', CLINICAL ENDOCRINOLOGY, Adelaide, AUSTRALIA (2016) | ||||
2012 | Sutherland JM, Fraser BA, Siddall NA, Hime GR, Davidson T-L, Koopman P, McLaughlin EA, 'Musashi-2: an essential regulator of DNA recombination and repair', Abstracts. The Annual Scientific Meeting of the Endocrine Society of Australia and the Society for Reproductive Biology 2012, Gold Coast, QLD (2012) [E3] | ||||
2010 | Sutherland JM, Keightley RA, Robker RL, Russell DL, McLaughlin EA, 'JAK/STAT Signalling in Folliculogenesis', Reproduction, Fertility and Development, Sydney (2010) [E3] | ||||
Show 2 more conferences |
Preprint (2 outputs)
Year | Citation | Altmetrics | Link | |||||
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2022 |
Frost ER, Taylor G, Boeing S, Galichet C, Baker MA, Sutherland JM, Lovell-Badge R, 'p27
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2020 |
Siddall NA, Casagranda F, Johanson TM, Dominado N, Heaney J, Sutherland JM, McLaughlin EA, 'MiMIC analysis reveals an isoform specific role for
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Grants and Funding
Summary
Number of grants | 19 |
---|---|
Total funding | $1,131,456 |
Click on a grant title below to expand the full details for that specific grant.
Highlighted grants and funding
The role of primordial follicle activation in premature ovarian failure$348,023
Funding body: NHMRC (National Health & Medical Research Council)
Funding body | NHMRC (National Health & Medical Research Council) |
---|---|
Project Team | Doctor Jessie Sutherland |
Scheme | Early Career Fellowships |
Role | Lead |
Funding Start | 2017 |
Funding Finish | 2021 |
GNo | G1600095 |
Type Of Funding | C1100 - Aust Competitive - NHMRC |
Category | 1100 |
UON | Y |
20232 grants / $19,312
Empowering adolescents to improve sexual health outcomes$14,767
Funding body: Hunter New England Local Health District
Funding body | Hunter New England Local Health District |
---|---|
Project Team | Doctor Jessie Sutherland, Dr Rajyalakshmi Kasi, Doctor Emmalee Ford |
Scheme | John Hunter Hospital Charitable Trust Grant |
Role | Lead |
Funding Start | 2023 |
Funding Finish | 2023 |
GNo | G2300395 |
Type Of Funding | C2400 – Aust StateTerritoryLocal – Other |
Category | 2400 |
UON | Y |
A Fertile Future$4,545
Funding body: CSIRO - Commonwealth Scientific and Industrial Research Organisation
Funding body | CSIRO - Commonwealth Scientific and Industrial Research Organisation |
---|---|
Project Team | Doctor Jessie Sutherland, Doctor Emmalee Ford |
Scheme | ON Prime |
Role | Lead |
Funding Start | 2023 |
Funding Finish | 2023 |
GNo | G2300405 |
Type Of Funding | C2100 - Aust Commonwealth – Own Purpose |
Category | 2100 |
UON | Y |
20221 grants / $414,113
Determining the regulation of ovary development with single cell sequencing$414,113
Funding body: ARC (Australian Research Council)
Funding body | ARC (Australian Research Council) |
---|---|
Project Team | Doctor Jessie Sutherland |
Scheme | Discovery Early Career Researcher Award (DECRA) |
Role | Lead |
Funding Start | 2022 |
Funding Finish | 2024 |
GNo | G2001133 |
Type Of Funding | C1200 - Aust Competitive - ARC |
Category | 1200 |
UON | Y |
20213 grants / $66,094
Improving fertility awareness and knowledge in young people$28,094
Funding body: Hunter Medical Research Institute
Funding body | Hunter Medical Research Institute |
---|---|
Project Team | Doctor Jessie Sutherland, Doctor Catherine Chojenta, Doctor Sarah Delforce, Doctor Emmalee Ford, Dr Sally Sweeney |
Scheme | Research Grant |
Role | Lead |
Funding Start | 2021 |
Funding Finish | 2022 |
GNo | G2101106 |
Type Of Funding | C3300 – Aust Philanthropy |
Category | 3300 |
UON | Y |
The role of prorenin in preparing the uterus for pregnancy$28,000
Funding body: Hunter Medical Research Institute
Funding body | Hunter Medical Research Institute |
---|---|
Project Team | Doctor Sarah Delforce, Doctor Jessie Sutherland, Professor Kirsty Pringle |
Scheme | Research Grant |
Role | Investigator |
Funding Start | 2021 |
Funding Finish | 2021 |
GNo | G2100201 |
Type Of Funding | C3300 – Aust Philanthropy |
Category | 3300 |
UON | Y |
Cooper Surgical Support for: Fertility Education in High School Project$10,000
Funding body: Origio Australasia Pty Ltd
Funding body | Origio Australasia Pty Ltd |
---|---|
Project Team | Doctor Jessie Sutherland, Doctor Emmalee Ford |
Scheme | Research Grant |
Role | Lead |
Funding Start | 2021 |
Funding Finish | 2021 |
GNo | G2001205 |
Type Of Funding | C3100 – Aust For Profit |
Category | 3100 |
UON | Y |
20191 grants / $4,982
COUNTESS II IF$4,982
Funding body: Hunter Medical Research Institute
Funding body | Hunter Medical Research Institute |
---|---|
Project Team | Doctor Jessie Sutherland, Doctor Elizabeth Bromfield |
Scheme | Early and Mid-Career Equipment Grant |
Role | Lead |
Funding Start | 2019 |
Funding Finish | 2019 |
GNo | G1900113 |
Type Of Funding | C3300 – Aust Philanthropy |
Category | 3300 |
UON | Y |
20181 grants / $20,000
Improving the diagnosis of women at risk of premature menopause$20,000
Funding body: Hunter Medical Research Institute
Funding body | Hunter Medical Research Institute |
---|---|
Project Team | Doctor Jessie Sutherland |
Scheme | Project Grant |
Role | Lead |
Funding Start | 2018 |
Funding Finish | 2018 |
GNo | G1801335 |
Type Of Funding | C3300 – Aust Philanthropy |
Category | 3300 |
UON | Y |
20176 grants / $421,147
The role of primordial follicle activation in premature ovarian failure$348,023
Funding body: NHMRC (National Health & Medical Research Council)
Funding body | NHMRC (National Health & Medical Research Council) |
---|---|
Project Team | Doctor Jessie Sutherland |
Scheme | Early Career Fellowships |
Role | Lead |
Funding Start | 2017 |
Funding Finish | 2021 |
GNo | G1600095 |
Type Of Funding | C1100 - Aust Competitive - NHMRC |
Category | 1100 |
UON | Y |
2017 UON Researcher Equipment Grant$50,000
Funding body: The University of Newcastle
Funding body | The University of Newcastle |
---|---|
Project Team | Dr Jessie Sutherland, Dr Zamira Gibb, |
Scheme | 2017 UON Researcher Equipment Grants Scheme |
Role | Investigator |
Funding Start | 2017 |
Funding Finish | 2017 |
GNo | |
Type Of Funding | Internal |
Category | INTE |
UON | N |
DVCRI Research Support for Early Career Fellow (ECF17)$17,624
Funding body: University of Newcastle
Funding body | University of Newcastle |
---|---|
Project Team | Doctor Jessie Sutherland |
Scheme | NHMRC ECF Support |
Role | Lead |
Funding Start | 2017 |
Funding Finish | 2022 |
GNo | G1700312 |
Type Of Funding | Internal |
Category | INTE |
UON | Y |
World Congress in Reproductive Biology 2017 ECR Travel Grant $2,000
Funding body: Australian Society for Reproductive Biology
Funding body | Australian Society for Reproductive Biology |
---|---|
Project Team | Dr Jessie Sutherland |
Scheme | World Congress in Reproductive Biology 2017 ECR Travel Grant |
Role | Lead |
Funding Start | 2017 |
Funding Finish | 2017 |
GNo | |
Type Of Funding | External |
Category | EXTE |
UON | N |
Howard Hughes Medical Institute Award$2,000
Funding body: Howard Hughes Medical Institute
Funding body | Howard Hughes Medical Institute |
---|---|
Project Team | Dr Jessie Sutherland |
Scheme | Cold Spring Harbor Mouse Development Stem Cells and Cancer Training Course |
Role | Lead |
Funding Start | 2017 |
Funding Finish | 2017 |
GNo | |
Type Of Funding | C3220 - International Philanthropy |
Category | 3220 |
UON | N |
Society for Reproductive Biology Early Career Researcher Collaborative Research Travel Award$1,500
Funding body: Australian Society for Reproductive Biology
Funding body | Australian Society for Reproductive Biology |
---|---|
Project Team | Dr Jessie Sutherland, Dr Kylie Dunning |
Scheme | Early Career Researcher Collaborative Research Travel Award |
Role | Lead |
Funding Start | 2017 |
Funding Finish | 2018 |
GNo | |
Type Of Funding | External |
Category | EXTE |
UON | N |
20163 grants / $93,934
The University of Newcastle Early Career Researcher Equipment Grant$50,000
Funding body: Univeristy of Newcastle
Funding body | Univeristy of Newcastle |
---|---|
Project Team | Dr Jessie Sutherland, Dr Kate Redgrove |
Scheme | The University of Newcastle Early Career Researcher Equipment Grant Scheme 2016 |
Role | Lead |
Funding Start | 2016 |
Funding Finish | 2017 |
GNo | |
Type Of Funding | Internal |
Category | INTE |
UON | N |
Beckman Coulter Optima Max-TL, Benchtop Ultra Centrifuge, TLA-110 Fixed-Angle Rotor Package, TLS-55 Swinging Bucket Rotor Package$40,934
Funding body: NHMRC (National Health & Medical Research Council)
Funding body | NHMRC (National Health & Medical Research Council) |
---|---|
Project Team | Professor Brett Nixon, Distinguished Emeritus Professor John Aitken, Professor Eileen McLaughlin, Associate Professor Mark Baker, Doctor Jessie Sutherland, Doctor Elizabeth Bromfield |
Scheme | Equipment Grant |
Role | Investigator |
Funding Start | 2016 |
Funding Finish | 2016 |
GNo | G1601314 |
Type Of Funding | C2200 - Aust Commonwealth – Other |
Category | 2200 |
UON | Y |
2016 Equal Futures Award$3,000
Funding body: Hunter Medical Research Institute
Funding body | Hunter Medical Research Institute |
---|---|
Project Team | Doctor Jessie Sutherland |
Scheme | Equal Futures Award |
Role | Lead |
Funding Start | 2016 |
Funding Finish | 2017 |
GNo | G1601263 |
Type Of Funding | Grant - Aust Non Government |
Category | 3AFG |
UON | Y |
20151 grants / $20,200
Understanding female fertility: The importance of cytokine signalling in premature menopause$20,200
Funding body: Hunter Medical Research Institute
Funding body | Hunter Medical Research Institute |
---|---|
Project Team | Doctor Jessie Sutherland |
Scheme | Project Grant |
Role | Lead |
Funding Start | 2015 |
Funding Finish | 2015 |
GNo | G1501433 |
Type Of Funding | Grant - Aust Non Government |
Category | 3AFG |
UON | Y |
20141 grants / $71,674
JuLI Stage $71,674
Funding body: NHMRC (National Health & Medical Research Council)
Funding body | NHMRC (National Health & Medical Research Council) |
---|---|
Project Team | Professor Pradeep Tanwar, Professor Eileen McLaughlin, Emeritus Professor Robin Callister, Professor Xu Dong Zhang, Professor Murray Cairns, Professor Brett Nixon, Professor Hubert Hondermarck, Associate Professor Phillip Dickson, Associate Professor Nikki Verrills, Professor Matt Dun, Doctor Jessie Sutherland, Doctor Janani Kumar, Professor Jay Horvat, Associate Professor Susan Hua, Prof LIZ Milward, Associate Professor Estelle Sontag, Professor Dirk Van Helden, Doctor Janet Bristow, Doctor Jean-Marie Sontag |
Scheme | Equipment Grant |
Role | Investigator |
Funding Start | 2014 |
Funding Finish | 2014 |
GNo | G1500860 |
Type Of Funding | Other Public Sector - Commonwealth |
Category | 2OPC |
UON | Y |
Research Supervision
Number of supervisions
Current Supervision
Commenced | Level of Study | Research Title | Program | Supervisor Type |
---|---|---|---|---|
2023 | PhD | Determining the Regulation of Ovary Development with Single Cell Sequencing | PhD (Medical Biochemistry), College of Health, Medicine and Wellbeing, The University of Newcastle | Principal Supervisor |
2022 | PhD | Causes And Consequences Of Placental Angiotensin Converting Enzyme 2 (ACE2) Deficiency For Fetal Growth | PhD (Medical Biochemistry), College of Health, Medicine and Wellbeing, The University of Newcastle | Co-Supervisor |
2019 | PhD | The Role of Autophagy in Oocyte Development and Ageing | PhD (Medical Biochemistry), College of Health, Medicine and Wellbeing, The University of Newcastle | Principal Supervisor |
Past Supervision
Year | Level of Study | Research Title | Program | Supervisor Type |
---|---|---|---|---|
2021 | PhD | A Diversified Approach to Improving Fertility Outcomes: Understanding Women’s Fertility Knowledge through Apps and Primordial Follicle Activation in Granulosa Cells | PhD (Medical Biochemistry), College of Health, Medicine and Wellbeing, The University of Newcastle | Principal Supervisor |
2021 | PhD | Regulation of Sperm Function and Oxidative Stress by Lipoxygenase Enzymes | PhD (Biological Sciences), College of Engineering, Science and Environment, The University of Newcastle | Co-Supervisor |
2021 | PhD | The Role of Cell Cycle Regulation in Granulosa Cells during Mouse Ovarian Development | PhD (Medical Biochemistry), College of Health, Medicine and Wellbeing, The University of Newcastle | Principal Supervisor |
News
News • 5 Nov 2021
2021 HMRI Awards for Research Excellence announced
The HMRI Awards are a celebration of the outstanding efforts and achievements of individuals and teams who drive and support the opportunities that health and medical research bring to the wellbeing of our community.
News • 17 Aug 2021
Funding success supports early career research translate to real-world
Five outstanding early career researchers have been successful in securing more than $2 million in the Australian Research Council’s Discovery Early Career Researcher Award (DECRA) scheme.
News • 27 Oct 2016
UON awarded over $5.6 million in NHMRC funding
The University of Newcastle (UON) is delighted to announce the following successful researchers in the latest round of National Health and Medical Research Council funding. With the help of this funding, our researchers aim to tackle a range of health-related issues that impact our communities.
Dr Jessie Sutherland
Position
ARC DECRA
HMRI Pregnancy and Reproduction Program Priority Research Centre for Reproductive Science
School of Biomedical Sciences and Pharmacy
College of Health, Medicine and Wellbeing
Contact Details
jessie.sutherland@newcastle.edu.au | |
Phone | (02) 4913 8735 |
Office
Room | LS2.35 |
---|---|
Building | Life Science Building |
Location | Callaghan University Drive Callaghan, NSW 2308 Australia |