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Associate Professor Martin Veysey

Associate Professor

School of Medicine and Public Health

Career Summary

Biography

I completed my undergraduate training at the UMDS, Guys and St Thomas’ Hospitals, University of London in 1991 and my Internship at Guys and Lewisham Hospitals during the following 12 months. I then moved to South Wales where I spent the next two years as a basic trainee at the Princess of Wales Hospital, Bridgend.

In mid 1994, I gained membership to the Royal College of Physicians of London. Later that year, I took up a research position at Guys Hospital and completed my MD thesis entitled, “The role of intestinal transit in the pathogenesis of octreotide-induced gallstones”, awarded by the University of London in January 2001. I commenced Advanced Training in Gastroenterology and General Internal Medicine in the South East Thames Rotation in 1997. After two years at Greenwich District Hospital, I spent the next 18 months at Kings College Hospital working as the Registrar to the Liver Unit. I spent the next 12 months in Australia as a Medical Registrar at Gosford Hospital, before completing my training at St Thomas Hospital, London, in 2002.

I migrated to Australia in 2003 with my family, upon obtaining my current position, and have lived and worked on the Central Coast since that time. I have significant experience in the management of the full range of gastroenterological, liver and general medical conditions. My clinical and research interests include medical education, molecular nutrition, colorectal cancer and luminal gastrointestinal disease. I am Director of the Teaching & Research Unit at Gosford Hospital and Clinical Dean of the Central Coast Clinical School of the Joint Medical Program, coordinating the clinical placements of 80 year 4 and 5 medical students.

Research Expertise
Having completed a MD research program in the UK between 1994 and 1997 and developed an international reputation as a bile acid researcher, I came to the University in 2003 after five years of clinical training with no active research activity. Since that time, I have assisted in the development of a completely novel research program with my colleagues in the Nutrition, Food and Health Research Group at the Ourimbah Campus investigating the role of B vitamins in degenerative illnesses, specifically vascular disease and cancer. I have co-supervised, and currently have, a number of PhD students working with me. More recently, I have negotiated with a number of industry partners to put together and lead a multidisciplinary team across three universities to investigate the health impacts of retirement village living, using B vitamin metabolism and vascular health as clinical markers. This work was supported by the ARC and is currently being evaluated.

Teaching Expertise
I have extensive experience in both undergraduate and postgraduate education. Through my role as Associate Professor at the University I have a significant tertiary teaching load and administrative responsibility. I am the Year Chair for years 1 and 2 of the Joint Medicial Program. I am also Course Coordinator for two first year courses (MEDI1011 Introduction to professional practice and MEDI1014 Professional Practice 1). Through my role as the Clinical Dean of the Central Coast Clinical School, I coordinate and teach Medicine to 80 Year 4 and 5 students at Gosford. I am involved in the JMP committee, assessment and admissions sub-committees and I am active member of the Medical Education Unit. I have also been involved in AMC and FRACP training. I have organised the FRACP Adult Medicine examination at Gosford, initially as a Regional Examiner, and in the last seven years as a Member of National and Senior Examining Panels. Since 2003, I have been an active member of the Central Coast Health Service General Clinical Training Committee monitoring and supporting the education of PGY1 and 2 doctors. Through the roles outlined above, I have developed a clear knowledge and understanding of modern educational principles including adult learning, problem based and self directed learning, e-learning and the delivery of educational courses over a wide footprint. I have completed a Graduate Certificate in the Practice of Tertiary Teaching with the University of Newcastle and a Master of Clinical Education with Flinders University, South Australia.

Administrative Expertise
Current University Administrative Roles Year 1/2 Chair, JMP (since 2011) Course coordinator MEDI 1011 (since 2011), MEDI 1014 JMP (since 2005) Clinical Dean, Central Coast Clinical School, JMP (since 2008) Current Committee Experience University of Newcastle, Faculty of Health, Executive Committee (since 2004) University of Newcastle, School of Medicine and Public Health, Executive Committee (since 2005) University of Newcastle, School of Medicine and Public Health, JMP Teaching & Learning Committee (since 2003), JMP Assessment Committee (since 2010), JMP Admission Committee (since 2011), JMP Medical Education Unit (since 2011), JMP CDWP (since 2014, Chair Phase 1 JMP-MD) Central Coast Local Health District, Research Advisory Committee (since 2003, Chair 2005-11, since 2014) Central Coast Local Health District, Drugs & Therapeutics Committee (since 2005) Central Coast Local Health District, MDAAC (since 2006) Royal Australasian College of Physicians, OTP Committee (since 2015, Chair) Royal Australasian College of Physicians, College Education Committee (since 2014) Previous Committee Experience Postgraduate Medical Council, NSW, Core Curriculum Implementation Committee (2005-06) Northern Sydney Central Coast Health, Human Research Ethics Committee (2003-08, Deputy Chair 2006-08) Northern Sydney Central Coast Health, Area Research Advisory Committee (2005-08) Central Coast Local Health District, Clinical Council (2005-13) University of Newcastle, School of Medicine and Public Health, Research Advisory Committee (2006-10) University of Newcastle, Research Committee (2013-14) Northern Sydney Central Coast Health, Health Technology Evaluation Committee (2006-10, Co-Chair 2007-10) Royal Australasian College of Physicians, NSW State Committee (2006-12, co-chair 2006-12) Royal Australasian College of Physicians, Adult Medicine OTP Sub-Committee (2008-15, AON Lead Fellow 2009-12, Chair 2012-15 ) Royal Australasian College of Physicians, Adult Medicine Division Education Committee (2012-15) GMCT Gastroenterology Executive Committee (2006-13) Chairman, Gosford Hospital Medical Staff Council (2009-13) Other Relevant Administrative Experience Medical Coordinator for the Rotary Bowelscan Faecal Occult Blood Screening Program (Northern Sydney and Central Coast Regions) (since 2003) Member of the AMC Database for Accreditation Activities (since 2005) Regional Examiner for the Royal Australasian College of Physicians (since 2004) Member of National and Senior Examining Panels for the Royal Australasian College of Physicians (since 2006) Medical Coordinator for the Rotary Bowelscan Faecal Occult Blood Screening Program (Northern Sydney and Central Coast Regions) (since 2003) Chairman, Gosford Hospital Medical Staff Council (2009-13) Member of Supervision Policy Development Working Group for Royal Australasian College of Physicians (2013-14) Chair of Selection into Training Policy Development Working Group for the Royal Australasian College of Physicians (since 2014) Chair of Curriculum Advisory Group for Royal Australasian College of Physicians (since 2014)



Qualifications

  • PhD (Medicine), University of London
  • Bachelor of Medicine & Surgery, University of London
  • Diploma of Royal College of Physicians, Royal College of Physicians - London

Keywords

  • B vitamins
  • Colorectal cancer
  • Degenerative disease
  • Gastroenterology
  • Healthy ageing
  • Medical Education
  • Medicine
  • Molecular nutrition
  • Nutritional genetics
  • Supervision

Fields of Research

Code Description Percentage
060199 Biochemistry and Cell Biology not elsewhere classified 25
110399 Clinical Sciences not elsewhere classified 25
060499 Genetics not elsewhere classified 50

Professional Experience

UON Appointment

Title Organisation / Department
Associate Professor University of Newcastle
School of Medicine and Public Health
Australia

Academic appointment

Dates Title Organisation / Department
1/03/2015 -  Chair of OTP Committee Royal Australasian College of Physicians
1/11/2014 -  Chair of Curriculum Advisory Group Royal Australasian College of Physicians
1/01/2014 -  Chair of Selection into Training Working Group Royal Australasian College of Physicians
1/02/2003 -  Clinical Academic Central Coast Local Health District
Teaching & Research Unit
Edit

Publications

For publications that are currently unpublished or in-press, details are shown in italics.


Journal article (71 outputs)

Year Citation Altmetrics Link
2015 Lucock M, Yates Z, Martin C, Choi J-H, Beckett E, Boyd L, et al., 'Methylation diet and methyl group genetics in risk for adenomatous polyp occurrence', BBA Clinical, 3 107-112 (2015)

Purpose: The aim of this study is to explore whether a methylation diet influences risk for adenomatous polyps (AP) either independently, or interactively with one-carbon metaboli... [more]

Purpose: The aim of this study is to explore whether a methylation diet influences risk for adenomatous polyps (AP) either independently, or interactively with one-carbon metabolism-dependent gene variants, and whether such a diet modifies blood homocysteine, a biochemical phenotype closely related to the phenomenon of methylation. Methods: 249 subjects were examined using selective fluorescence, PCR and food frequency questionnaire to determine homocysteine, nine methylation-related gene polymorphisms, dietary methionine, 5-methyltetrahydrofolate, vitamins B6 and B12. Results: 1). Both dietary methionine and 5-methyltetrahydrofolate intake are significantly associated with plasma homocysteine. 2). Dietary methionine is related to AP risk in 2R3R-TS wildtype subjects, while dietary B12 is similarly related to this phenotype in individuals heterozygous for C1420T-SHMT, A2756G-MS and 844ins68-CBS, and in those recessive for 2R3R-TS. 3). Dietary methionine has a marginal influence on plasma homocysteine level in C1420T-SHMT heterozygotes, while B6 exhibits the same effect on homocysteine in C776G-TCN2 homozygote recessive subjects. Natural 5-methyltetrahydrofolate intake is interesting: Wildtype A1298C-MTHFR, heterozygote C677T-MTHFR, wildtype A2756G-MS and recessive A66G-MSR individuals all show a significant reciprocal association with homocysteine. 4). Stepwise regression of all genotypes to predict risk for AP indicated A2756G-MS and A66G-MSR to be most relevant (p= 0.0176 and 0.0408 respectively). Results were corrected for age and gender. Conclusion: A methylation diet influences methyl group synthesis in the regulation of blood homocysteine level, and is modulated by genetic interactions. Methylation-related nutrients also interact with key genes to modify risk of AP, a precursor of colorectal cancer. Independent of diet, two methylation-related genes (A2756G-MS and A66G-MSR) were directly associated with AP occurrence.

DOI 10.1016/j.bbacli.2014.11.005
Citations Scopus - 1
Co-authors Mark Lucock, Zoe Yates
2015 Siow WH, Hawken G, Russell A, Singh S, Hampe T, Veysey M, 'Education and imaging. Gastrointestinal: multiple inflammatory myoglandular polyps in a single patient.', J Gastroenterol Hepatol, 30 231 (2015)
DOI 10.1111/jgh.12822
2015 Choi JH, Yates Z, Martin C, Boyd L, Ng X, Skinner V, et al., 'Genetic Variation in Glutamate Carboxypeptidase II and Interaction with Dietary Natural Vitamin C May Predict Risk for Adenomatous Polyp Occurrence.', Asian Pac J Cancer Prev, 16 4383-4386 (2015)
Co-authors Mark Lucock, Zoe Yates
2015 Beckett EL, Martin C, Choi JH, King K, Niblett S, Boyd L, et al., 'Folate status, folate-related genes and serum miR-21 expression: Implications for miR-21 as a biomarker', BBA Clinical, 4 45-51 (2015)

Background: Free circulating microRNA (miRNA) in serum may be valuable biomarkers for disease diagnosis and prognosis. miR-21, the archetypal oncogenic miRNA, has been proposed as... [more]

Background: Free circulating microRNA (miRNA) in serum may be valuable biomarkers for disease diagnosis and prognosis. miR-21, the archetypal oncogenic miRNA, has been proposed as a biomarker for colorectal cancer and its benign precursor, adenomatous polyps. However, it is now becoming clear that circulating miRNA profiles may be sensitive to lifestyle and environmental influences. Dietary components involved in one-carbon metabolism are particularly well placed to modulate miRNA expression through an influence on DNA methylation pathways. Methods: We investigated the role of methyl group donors (folate, B12, cysteine, homocysteine), polymorphisms of the enzymes of one-carbon metabolism, and serum miR-21 expression in a primary case-control cohort (colonoscopy confirmed adenomatous colon polyps vs controls; n. =. 253) and a secondary cross-sectional cohort (over 65s; n. =. 649). The relationships between these parameters and serum miR-21 levels were assessed, stratified by gender. Conclusions: Serum miR-21 expression was related to occurrence of adenomatous polyps in females, but not males. Folate levels and MTHFR-C677T genotype was associated with miR-21 expression in both genders. Additionally, DHFR-19 del and MSR-A66G were associated with miR-21 expression in females and males, respectively. Stimulation with excess folate increased expression of miR-21 in colon cancer cell lines. General significance: This study demonstrates that serum miR-21 expression correlates with folate status and related genetic status. This may have consequences for the proposed use of miR-21 as a colorectal cancer biomarker.

DOI 10.1016/j.bbacli.2015.06.006
Co-authors Zoe Yates, Mark Lucock
2015 Lucock M, Jones P, Martin C, Beckett E, Yates Z, Furst J, Veysey M, 'Vitamin D: Beyond Metabolism.', J Evid Based Complementary Altern Med, (2015)
DOI 10.1177/2156587215580491
Co-authors Zoe Yates, Mark Lucock, John Furst
2015 Duvivier R, Kelly B, Veysey M, 'Selection and study performance', Medical Education, 49 638-639 (2015)
DOI 10.1111/medu.12691
Citations Scopus - 1Web of Science - 1
Co-authors Robbert Duvivier, Brian Kelly
2014 Beckett EL, Yates Z, Veysey M, Duesing K, Lucock M, 'The role of vitamins and minerals in modulating the expression of microRNA.', Nutr Res Rev, 27 94-106 (2014) [C1]
DOI 10.1017/S0954422414000043
Co-authors Mark Lucock, Zoe Yates
2014 Beckett EL, Martin C, Duesing K, Jones P, Furst J, Yates Z, et al., 'Vitamin D Receptor Genotype Modulates the Correlation between Vitamin D and Circulating Levels of let-7a/b and Vitamin D Intake in an Elderly Cohort.', J Nutrigenet Nutrigenomics, 7 264-273 (2014)
DOI 10.1159/000381676
Co-authors John Furst, Mark Lucock, Zoe Yates
2014 Beckett EL, Yates Z, Veysey M, Duesing K, Lucock M, 'The role of vitamins and minerals in modulating the expression of microRNA', Nutrition Research Reviews, 27 94-106 (2014) [C1]

A growing number of studies in recent years have highlighted the importance of molecular nutrition as a potential determinant of health and disease. In particular, the ability of ... [more]

A growing number of studies in recent years have highlighted the importance of molecular nutrition as a potential determinant of health and disease. In particular, the ability of micronutrients to regulate the final expression of gene products via modulation of transcription and translation is now being recognised. Modulation of microRNA (miRNA) by nutrients is one pathway by which nutrition may mediate gene expression. MiRNA, a class of non-coding RNA, can directly regulate gene expression post-transcriptionally. In addition, miRNA are able to indirectly influence gene expression potential at the transcriptional level via modulation of the function of components of the epigenetic machinery (DNA methylation and histone modifications). These mechanisms interact to form a complex, bi-directional regulatory circuit modulating gene expression. Disease-specific miRNA profiles have been identified in multiple disease states, including those with known dietary risk factors. Therefore, the role that nutritional components, in particular, vitamins and minerals, play in the modulation of miRNA profiles, and consequently health and disease, is increasingly being investigated, and as such is a timely subject for review. The recently posited potential for viable exogenous miRNA to enter human blood circulation from food sources adds another interesting dimension to the potential for dietary miRNA to contribute to gene modulation. © 2014 The Authors.

DOI 10.1017/S0954422414000043
Citations Scopus - 2Web of Science - 1
Co-authors Zoe Yates, Mark Lucock
2014 Beckett EL, Yates Z, Veysey M, Duesing K, Lucock M, 'The role of vitamins and minerals in modulating the expression of microRNA', Nutrition Research Reviews, 27 94-106 (2014)
DOI 10.1017/S0954422414000043
Co-authors Mark Lucock, Zoe Yates
2014 Beckett EL, Yates Z, Veysey M, Duesing K, Lucock M, 'The role of vitamins and minerals in modulating the expression of microRNA', Nutrition Research Reviews, 96 (2014)

A growing number of studies in recent years have highlighted the importance of molecular nutrition as a potential determinant of health and disease. In particular, the ability of ... [more]

A growing number of studies in recent years have highlighted the importance of molecular nutrition as a potential determinant of health and disease. In particular, the ability of micronutrients to regulate the final expression of gene products via modulation of transcription and translation is now being recognised. Modulation of microRNA (miRNA) by nutrients is one pathway by which nutrition may mediate gene expression. miRNA, a class of non-coding RNA, can directly regulate gene expression post-transcriptionally. In addition, miRNA are able to indirectly influence gene expression potential at the transcriptional level via modulation of the function of components of the epigenetic machinery (DNA methylation and histone modifications). These mechanisms interact to form a complex, bi-directional regulatory circuit modulating gene expression. Disease-specific miRNA profiles have been identified in multiple disease states, including those with known dietary risk factors. Therefore, the role that nutritional components, in particular, vitamins and minerals, play in the modulation of miRNA profiles, and consequently health and disease, is increasingly being investigated, and as such is a timely subject for review. The recently posited potential for viable exogenous miRNA to enter human blood circulation from food sources adds another interesting dimension to the potential for dietary miRNA to contribute to gene modulation.

DOI 10.1017/S0954422414000043
Citations Scopus - 1
Co-authors Mark Lucock, Zoe Yates
2014 Beckett EL, Martin C, Duesing K, Jones P, Furst J, Yates Z, et al., 'Vitamin D receptor genotype modulates the correlation between vitamin D and circulating levels of let-7a/b and vitamin D intake in an elderly cohort', Journal of Nutrigenetics and Nutrigenomics, 7 264-273 (2014)

Background and Aims: Circulating microRNAs (miRNAs) are linked to disease and are potential biomarkers. Vitamin D may modulate miRNA profiles, and vitamin D status has been linked... [more]

Background and Aims: Circulating microRNAs (miRNAs) are linked to disease and are potential biomarkers. Vitamin D may modulate miRNA profiles, and vitamin D status has been linked to risk of disease, including cardiovascular disease and cancers. We hypothesise that genotypic variance influences these relationships. We examined the correlations between vitamin D intake and circulating levels of the miRNAs let-7a/b, and the involvement of two common vitamin D receptor (VDR) polymorphisms, BsmI and ApaI. Methods: Two hundred participants completed food frequency and supplement questionnaires, and were assayed for circulating let-7b expression by qPCR. Polymorphisms were detected using restriction fragment length polymorphism-PCR. Results: let-7b expression negatively correlated with vitamin D intake (rs = -0.20, p = 0.005). The magnitude and direction of correlation were maintained in the presence of the BsmI restriction site (rs = -0.27, p = 0.0005). However, in the absence of BsmI restriction site, the direction of the correlation was reversed (rs = +0.319, p = 0.0497). These correlations were significantly different (z-score = 2.64, p = 0.0085). The correlation between vitamin D intake and let-7a was only significant in those without the ApaI restriction site. Conclusions: The correlation between vitamin D intake and let-7a/b expression in this cohort varies with VDR genotype. This study highlights the importance of considering underlying genotypic variance in miRNA expression studies and in nutritional epigenetics generally.

DOI 10.1159/000381676
Citations Scopus - 2
Co-authors Mark Lucock, Zoe Yates, John Furst
2014 Beckett EL, Martin C, Yates Z, Veysey M, Duesing K, Lucock M, 'Bitter taste genetics-the relationship to tasting, liking, consumption and health', Food and Function, 5 3040-3054 (2014) [C1]

Bitter is the most complex of human tastes, and is arguably the most important. Aversion to bitter taste is important for detecting toxic compounds in food; however, many benefici... [more]

Bitter is the most complex of human tastes, and is arguably the most important. Aversion to bitter taste is important for detecting toxic compounds in food; however, many beneficial nutrients also taste bitter and these may therefore also be avoided as a consequence of bitter taste. While many polymorphisms in TAS2R genes may result in phenotypic differences that influence the range and sensitivity of bitter compounds detected, the full extent to which individuals differ in their abilities to detect bitter compounds remains unknown. Simple logic suggests that taste phenotypes influence food preferences, intake and consequently health status. However, it is becoming clear that genetics only plays a partial role in predicting preference, intake and health outcomes, and the complex, pleiotropic relationships involved are yet to be fully elucidated. This journal is

DOI 10.1039/c4fo00539b
Co-authors Mark Lucock, Zoe Yates
2014 Lucock MD, Martin CE, Yates ZR, Veysey M, 'Diet and Our Genetic Legacy in the Recent Anthropocene: A Darwinian Perspective to Nutritional Health', Journal of Evidence-Based Complementary and Alternative Medicine, 19 68-83 (2014) [C1]

Nutrient-gene research tends to focus on human disease, although such interactions are often a by-product of our evolutionary heritage. This review explores health in this context... [more]

Nutrient-gene research tends to focus on human disease, although such interactions are often a by-product of our evolutionary heritage. This review explores health in this context, reframing genetic variation/epigenetic phenomena linked to diet in the framework of our recent evolutionary past. This "Darwinian/evolutionary medicine" approach examines how diet helped us evolve among primates and to adapt (or fail to adapt) our metabolome to specific environmental conditions leading to major diseases of civilization. This review presents updated evidence from a diet-gene perspective, portraying discord that exists with respect to health and our overall nutritional, cultural, and activity patterns. While Darwinian theory goes beyond nutritional considerations, a significant component within this concept does relate to nutrition and the mismatch between genes, modern diet, obesogenic lifestyle, and health outcomes. The review argues that nutritional sciences should expand knowledge on the evolutionary connection between food and disease, assimilating it into clinical training with greater prominence. © The Author(s) 2013.

DOI 10.1177/2156587213503345
Citations Scopus - 3
Co-authors Zoe Yates, Mark Lucock
2014 Choi J-H, Yates Z, Veysey M, Heo Y-R, Lucock M, 'Contemporary issues surrounding folic acid fortification initiatives', Preventive Nutrition and Food Science, 19 247-260 (2014) [C1]

The impact of folate on health and disease, particularly pregnancy complications and congenital malformations, has been extensively studied. Mandatory folic acid fortification the... [more]

The impact of folate on health and disease, particularly pregnancy complications and congenital malformations, has been extensively studied. Mandatory folic acid fortification therefore has been implemented in multiple countries, resulting in a reduction in the occurrence of neural tube defects. However, emerging evidence suggests increased folate intake may also be associated with unexpected adverse effects. This literature review focuses on contemporary issues of concern, and possible underlying mechanisms as well as giving consideration the future direction of mandatory folic acid fortification. Folate fortification has been associated with the presence of unmetabolized folic acid (PteGlu) in blood, masking of vitamin B12 deficiency, increased dosage for anti-cancer medication, photo-catalysis of PteGlu leading to potential genotoxicity, and a role in the pathoaetiology of colorectal cancer. Increased folate intake has also been associated with twin birth and insulin resistance in offspring, and altered epigenetic mechanisms of inheritance. Although limited data exists to elucidate potential mechanisms underlying these issues, elevated blood folate level due to the excess use of PteGlu without consideration of an individual's specific phenotypic traits (e.g. genetic background and undiagnosed disease) may be relevant. Additionally, the accumulation of unmetabolized PteGlu may lead to inhibition of dihydrofolate reductase and other enzymes. Concerns notwithstanding, folic acid fortification has achieved enormous advances in public health. It therefore seems prudent to target and carefully monitor high risk groups, and to conduct well focused further research to better understand and to minimize any risk of mandatory folic acid fortification.

DOI 10.3746/pnf.2014.19.4.247
Citations Scopus - 1
Co-authors Zoe Yates, Mark Lucock
2014 Lucock M, Yates Z, Martin C, Choi JH, Boyd L, Tang S, et al., 'Vitamin D, folate, and potential early lifecycle environmental origin of significant adult phenotypes.', Evolution, Medicine, and Public Health, 2014 69-91 (2014) [C1]
DOI 10.1093/emph/eou013
Co-authors Zoe Yates, Mark Lucock, Paul Roach, John Furst
2014 Beckett EL, Martin C, Yates Z, Veysey M, Duesing K, Lucock M, 'Bitter taste genetics--the relationship to tasting, liking, consumption and health.', Food Funct, 5 3040-3054 (2014) [C1]
DOI 10.1039/c4fo00539b
Co-authors Mark Lucock, Zoe Yates
2014 Martin CE, Veysey M, Yates Z, Lucock MD, 'Vitamin D: Genetics, Environment & Health', Food and Nutritional Disorders, 3 1-19 (2014) [C1]
DOI 10.4172/2324-9323.1000155
Co-authors Mark Lucock, Zoe Yates
2013 Lucock MD, Martin C, Boyd L, Naumovski N, Roach P, Yates Z, Veysey M, 'Response to 'calcium, phosphate and the risk of cardiovascular events and all-cause mortality in a population with stable coronary heart disease'', HEART, 99 349-350 (2013) [C3]
DOI 10.1136/heartjnl-2012-302480
Co-authors Zoe Yates, Paul Roach, Mark Lucock
2013 Lucock M, Yates Z, Boyd L, Naylor C, Choi J, Ng X, et al., 'Vitamin C-related nutrient-nutrient and nutrient-gene interactions that modify folate status', European Journal of Nutrition, 52 569-582 (2013) [C1]
DOI 10.1007/s00394-012-0359-8
Citations Scopus - 8Web of Science - 4
Co-authors Zoe Yates, Paul Roach, Mark Lucock
2013 Lucock M, Yates Z, Martin C, Choi J, Boyd L, Tang S, et al., 'Hydrogen sulphide-related thiol metabolism and nutrigenetics in relation to hypertension in an elderly population', Genes & Nutrition, 8 221-229 (2013) [C1]
DOI 10.1007/s12263-012-0317-3
Citations Scopus - 3Web of Science - 1
Co-authors Paul Roach, Zoe Yates, Mark Lucock
2011 Lucock MD, Ng X, Boyd L, Skinner VM, Wai R, Tang S, et al., 'TAS2R38 bitter taste genetics, dietary vitamin C, and both natural and synthetic dietary folic acid predict folate status, a key micronutrient in the pathoaetiology of adenomatous polyps', Food & Function, 2 457-465 (2011) [C1]
DOI 10.1039/c1fo10054h
Citations Scopus - 7Web of Science - 5
Co-authors Mark Lucock, Paul Roach, Zoe Yates
2010 Jootun NR, Cheah HP, Fernando SC, Munro WS, Veysey M, 'Heterotopic pancreas causing intussusception in a child', MEDICAL JOURNAL OF AUSTRALIA, 192 542-542 (2010) [C3]
2010 Naumovski N, Veysey M, Ng X, Boyd L, Dufficy L, Blades BL, et al., 'The folic acid endophenotype and depression in an elderly population', Journal of Nutrition, Health and Aging, 14 829-833 (2010) [C1]
DOI 10.1007/s12603-010-0135-5
Citations Scopus - 3Web of Science - 2
Co-authors Maureen Townley-Jones, Paul Roach, Zoe Yates, Mark Lucock
2009 Ng X, Boyd L, Dufficy L, Naumovski N, Blades BL, Travers C, et al., 'Folate nutritional genetics and risk for hypertension in an elderly population sample', Journal of Nutrigenetics and Nutrigenomics, 2 1-8 (2009) [C1]
DOI 10.1159/000160079
Citations Scopus - 12Web of Science - 9
Co-authors Zoe Yates, Maureen Townley-Jones, Paul Roach, Mark Lucock
2009 Blacklaws H, Veysey H, Skinner VM, Reid RS, Hawken G, Veysey M, 'Interferon treatment for chronic hepatitis C: A family impact study', Gastroenterology Nursing, 32 377-383 (2009) [C1]
DOI 10.1097/SGA.0b013e3181c10759
Citations Scopus - 3Web of Science - 4
2008 Ng X, Lucock M, Veysey M, 'Physicochemical effect of pH and antioxidants on mono- and triglutamate forms of 5-methyltetrahydrofolate, and evaluation of vitamin stability in human gastric juice: Implications for folate bioavailability (vol 106, pg 200, 2008)', FOOD CHEMISTRY, 110 1000-1000 (2008) [C3]
DOI 10.1016/j.foodchem.2008.04.001
Co-authors Mark Lucock
2008 Ng X, Lucock MD, Veysey MJ, 'Physicochemical effect of pH and antioxidants on mono- and triglutamate forms of 5-methyltetrahydrofolate, and evaluation of vitamin stability in human gastric juice: Implications for folate bioavailability', Food Chemistry, 106 200-210 (2008) [C1]
DOI 10.1016/j.foodchem.2007.05.057
Citations Scopus - 11Web of Science - 9
Co-authors Mark Lucock
2007 Lucock MD, Yates ZR, Ng X, Veysey MJ, Blades BL, Travers C, et al., 'Preliminary evidence for genetic selection of 677T-MTHFR by natural annual cycle of folate abundance', Journal of Nutrigenetics and Nutrigenomics, 1 24-29 (2007) [C1]
DOI 10.1159/000109872
Citations Scopus - 10Web of Science - 8
Co-authors Paul Roach, Zoe Yates, Mark Lucock
2006 Dufficy L, Naumovski N, Ng X, Blades BL, Yates ZR, Travers C, et al., 'G80A reduced folate carrier SNP influences the absorption and cellular translocation of dietary folate and its association with blood pressure in an elderly population', Life Sciences, 79 957-966 (2006) [C1]
DOI 10.1016/j.lfs.2006.05.009
Citations Scopus - 20Web of Science - 20
Co-authors Mark Lucock, Zoe Yates, Paul Roach
2006 Veysey M, King K, Walsh P, Vigenser B, Leijten W, 'What happens to older and younger people with a national bowel cancer screening program?', JOURNAL OF GASTROENTEROLOGY AND HEPATOLOGY, 21 A285-A285 (2006)
2006 Veysey M, Blacklaws H, Decker E, Chaussivert I, Mackender D, 'Nurse-led liver clinics: do they work?', JOURNAL OF GASTROENTEROLOGY AND HEPATOLOGY, 21 A319-A319 (2006)
2005 Lucock MD, Ng X, Veysey MJ, Yates ZR, 'Folic acid: An essential nutrient with added health benefits', Biologist, 52 21-27 (2005) [C1]
Citations Scopus - 2
Co-authors Zoe Yates, Mark Lucock
2005 Thomas LA, Veysey MJ, Murphy GM, Jones DR, French GL, Wass JA, Dowling RH, 'Octreotide-induced prolongation of colonic transit increases faecal anaerobic bacteria, bile acid metabolising enzymes, and serum deoxycholic acid in patients with acromegaly', Gut, 54 630-635 (2005) [C1]
DOI 10.1136/gut.2003.028431
Citations Scopus - 20Web of Science - 19
2005 Lipski PS, Veysey M, 'A prospective audit of major adverse drug reactions causing acute hospital admission of elderly patients', AUSTRALASIAN JOURNAL ON AGEING, 24 A44-A44 (2005)
2001 Veysey MJ, Thomas LA, Mallet A, Jenkins P, Besser P, Murphy GM, Dowling RH, 'Colonic transit influences deoxycholic acid kinetics', Gastroenterology, (2001) [C1]
Citations Scopus - 20Web of Science - 18
2001 Thomas LA, Veysey MJ, French G, Hylemon P, Murphy GM, Dowling RH, 'Bile acid metabolism by fresh human colonic contents: a comparison of caecal versus faecal samples', GUT, (2001) [C1]
Citations Scopus - 41Web of Science - 43
2001 Veysey MJ, Malcolm P, Mallet A, Jenkins P, Besser P, Wass JA, et al., 'The effects of cisapride on gallbladder emptying, intestinal transit and serum deoxycholate: a prospective randomised, double blind, placebo-controlled trial.', Gut, (2001) [C1]
Citations Scopus - 13Web of Science - 11
2001 Thomas LA, Veysey MJ, Murphy GM, Dowling RH, 'Influence of pH on the phase distribution of nascent deoxycholic acid in fresh human caecal aspirates', Am J Physiol, (2001) [C1]
Citations Scopus - 4Web of Science - 2
2000 Stellini M, Sanderson JD, Jenkins PJ, Fairclough PD, Besser GM, Veysey MJ, et al., 'A novel explanation for the high prevalence of colorectal adenomas and cancers seen in acromegaly.', GASTROENTEROLOGY, 118 A60-A60 (2000)
2000 Thomas LA, Veysey MJ, Bathgate T, King A, French G, Smeeton NC, et al., 'Mechanism for the transit-induced increase in colonic deoxycholic acid formation in cholesterol cholelithiasis', GASTROENTEROLOGY, 119 806-815 (2000)
DOI 10.1053/gast.2000.16495
Citations Scopus - 66Web of Science - 53
1999 Veysey MJ, Thomas LA, Mallet AI, Jenkins PJ, Besser GM, Wass JAH, et al., 'Prolonged large bowel transit increases serum deoxycholic acid: a risk factor for octreotide induced gallstones', GUT, 44 675-681 (1999)
Citations Scopus - 48Web of Science - 43
1999 Veysey MJ, Kamanyire R, Volans GN, 'Effects of drug overdose in television drama on presentations for self poisoning - Antifreeze poisonings give more insight into copycat behaviour', BRITISH MEDICAL JOURNAL, 319 1131-1131 (1999)
Citations Scopus - 10Web of Science - 9
1999 Veysey MJ, Kamanyire R, Volans GN, Pell J, Murdoch R, Davies SJC, et al., 'Effects of drug overdose in television drama on presentations for self poisoning (multiple letters) [1]', British Medical Journal, 319 1131-1132 (1999)
1998 Thomas LA, Bathgate T, Veysey MJ, King A, French GL, Murphy GM, Dowling RH, 'Increases in luminal pH promote the absorption of deoxycholic acid from the colon of patients with cholesterol gallbladder stones (GBS).', GASTROENTEROLOGY, 114 A545-A545 (1998)
DOI 10.1016/S0016-5085(98)82217-8
1998 Thomas LA, Bathgate T, Veysey MJ, Smeeton N, French GL, Murphy GM, Dowling RH, 'Is cholelithiasis an intestinal disease? Multivariate analyses of five pathogenetic factors.', GASTROENTEROLOGY, 114 A546-A546 (1998)
1998 Thomas LA, Bathgate T, Veysey MJ, Powrie J, Russell-Jones D, Wass JAH, et al., 'Roles of colonic transit, bacteriology, bile acid metabolizing enzymes and deoxycholic acid in the pathogenesis of gallstones and colorectal cancer in acromegalic patients before and during octreotide treatment.', GASTROENTEROLOGY, 114 A546-A546 (1998)
1998 Veysey MJ, Mallet A, Jenkins P, Besser GM, Murphy GM, Dowling RH, 'The effects of octreotide on the kinetics of deoxycholic and cholic acid.', GASTROENTEROLOGY, 114 A548-A548 (1998)
DOI 10.1016/S0016-5085(98)82229-4
1998 Veysey MJ, Mallet A, Jenkins P, Besser GM, Murphy GM, Dowling RH, 'Deoxycholic (DCA) and cholic acid (CA) kinetics in acromegalic patients treated with octreotide (OT)', GUT, 42 A11-A11 (1998)
Citations Web of Science - 2
1998 Thomas LA, Bathgate T, Veysey MJ, King A, French GL, Murphy GM, Dowling RH, 'In studies of deoxycholic acid (DCA) formation and absorption, is it important to obtain caecal, rather than faecal, samples?', GUT, 42 A11-A11 (1998)
1997 Jenkins PJ, Veysey MJ, Arraton SRD, Murphy GM, Dowling RH, Besser GM, Wass JAH, 'O-055. Acromegalic patients have increased large bowel transit time (LBTT) and serum deoxycholic acid levels (% DCA) after long-term octreotide therapy', Endocrinology and Metabolism, Supplement, 4 30-30 (1997)

Background: Octreotide (OT) therapy causes the formation of gallbladder stones (GBS) in up to 50% of acromegalic patients, due to an increase in the % DCA and the cholesterol satu... [more]

Background: Octreotide (OT) therapy causes the formation of gallbladder stones (GBS) in up to 50% of acromegalic patients, due to an increase in the % DCA and the cholesterol saturation of gallbladder bile, together with impairment of meal-stimulated gallbladder emptying. Prolongation of intestinal transit has been proposed as the mechanism for the increase in the % DCA, but our earlier unpaired studies did not show any significant effect of OT on large bowel transit - important since the colon is the site of DCA formation and absorption. Objective: To assess LBTT and biliary DCA before and during long-term (>3 months) OT treatment. Methods: LBTT was measured using radio-opaque marker shapes in 8 acromegalic patients (age range 22-69 yrs; 4 women). As there is an exchange, and ultimately an equilibrium, between bile acids in serum and bile, fasting serum levels of DCA provide an accurate estimate of biliary levels. The % DCA was therefore measured in fasting serum of 6 patients. Results: The mean LBTT increased from 42±SEM 4.3 h before to 55±5.1 h during OT treatment (p<0.0001) and the mean % DCA increased from 15±2.5% to 28±4.7% (p<0.05). There was a significant linear relationship between LBTT and % DCA (r = 0.87; p<0.0005). Conclusions: These results show that long-term OT therapy prolongs LBTT and that this prolongation is associated with an increase in % DCA in serum, and by implication in bile - factors important in the pathogenesis of OT-induced gallstones.

1997 Veysey MJ, Gathercole DJ, Mallet A, Jenkins P, Besser GM, Wass JAH, et al., 'Large bowel transit time influences deoxycholic acid input rate and pool size - Risk factors for octreotide-induced gallstones.', GASTROENTEROLOGY, 112 A525-A525 (1997)
Citations Web of Science - 5
1997 Veysey MJ, Gathercole DJ, Thomas LA, Jenkins P, Fairclough PD, Besser GM, et al., 'Fasting serum deoxycholic acid concentrations are increased in acromegalic patients with colorectal adenoma or carcinoma.', GASTROENTEROLOGY, 112 A674-A674 (1997)
1997 Veysey MJ, Gathercole DJ, Thomas LA, Jenkins P, Besser GM, Fairclough PD, et al., 'In acromegalics with colorectal adenomas or carcinomas, serum deoxycholic acid is increased', GUT, 40 TH194-TH194 (1997)
1997 Veysey MJ, Gathercole DJ, Mallet A, Jenkins P, Besser GM, Wass JAH, et al., 'The pathogenesis of octreotide-induced gallbladder stones - Prolonged large bowel transit increases deoxycholic acid (DCA) input rate and pool size', GUT, 40 F264-F264 (1997)
1997 Thomas LA, Veysey MJ, Murphy GM, Dowling RH, King A, French GR, 'Bile acid metabolising intestinal bacterial enzyme activity: A novel factor in cholesterol gallstone pathogenesis', GUT, 40 F266-F266 (1997)
Citations Web of Science - 1
1997 Thomas LA, Bathgate T, Veysey MJ, King A, French GL, Murphy GM, Dowling RH, 'Is cholelithiasis an intestinal disease?', GUT, 41 A2-A2 (1997)
Citations Web of Science - 6
1997 Dowling RH, Hussaini SH, Murphy GM, Pereira SP, Thomas LA, Veysey MJ, Wass JAH, 'Role of intestinal transit and DCA', GUT, 41 A31-A32 (1997)
1997 Thomas LA, Bathgate T, Veysey MJ, King A, French GR, Murphy GM, Dowling RH, 'Do changes in colonic luminal pH explain the increased proportions of serum and biliary deoxycholic acid seen in patients with cholesterol gallbladder stones (GBS)?', GUT, 41 A32-A33 (1997)
Citations Web of Science - 2
1997 Veysey MJ, Mills TD, Reynolds CR, Jenkins P, Besser GM, Dowling RH, '''Colonomegaly'' in acromegaly - A link with colorectal neoplasia?', GUT, 41 A121-A121 (1997)
Citations Web of Science - 1
1997 Veysey MJ, Jenkins P, Besser GM, Wass JAH, Dowling RH, 'The effect of cisapride on large bowel transit time: A randomised double-blind placebo-controlled study in four groups of individuals', GUT, 41 A123-A123 (1997)
Citations Web of Science - 1
1997 Thomas LA, Bathgate T, Veysey MJ, King A, French GR, Murphy GM, Dowling RH, 'In cholesterol gallstone disease, prolongation of large bowel transit time (LBTT) is associated with increases in the number of anaerobes and the activities of the deoxycholate-forming bacterial enzymes, in the right colon', GUT, 41 A241-A242 (1997)
1997 Thomas LA, Bathgate T, Veysey MJ, King A, French GL, Murphy GM, Dowling RH, 'Mechanism for increased deoxycholic acid (DCA) formation in acromegalic patients treated with octreotide (OT)', GUT, 41 A250-A251 (1997)
1997 Veysey MJ, Gathercole DJ, Jenkins P, Besser GM, Fairclough PD, Murphy GM, Dowling RH, 'Acromegalics with colorectal adenomas or carcinomas have increased serum deoxycholic acid (DCA) levels', GUT, 41 A251-A251 (1997)
Citations Web of Science - 1
1997 Behr ER, Veysey MJ, Berry D, Volans GN, 'Optimum dose of digoxin', LANCET, 349 1845-1845 (1997)
DOI 10.1016/S0140-6736(05)61735-7
1997 Zuccala G, Pedone C, Carosella L, Carbonin P, Bernabei R, Behr ER, et al., 'Optimum dose of digoxin (multiple letters) [9]', Lancet, 349 1845-1845 (1997)
Citations Scopus - 1
1997 Pereira SP, Veysey MJ, Kennedy C, Hussaini SH, Murphy GM, Dowling RH, 'Gallstone dissolution with oral bile acid therapy - Importance of pretreatment CT scanning and reasons for nonresponse', DIGESTIVE DISEASES AND SCIENCES, 42 1775-1782 (1997)
DOI 10.1023/A:1018834103873
Citations Scopus - 19Web of Science - 14
1997 Dowling RH, Veysey MJ, Pereira SP, Hussaini SH, Thomas LA, Wass JAH, Murphy GM, 'Role of intestinal transit in the pathogenesis of gallbladder stones', CANADIAN JOURNAL OF GASTROENTEROLOGY, 11 57-64 (1997)
Citations Web of Science - 32
1997 Dowling RH, Veysey MJ, Pereira SP, Hussaini SH, Thomas LA, Wass JAH, Murphy GM, 'Role of intestinal transit in the pathogenesis of gallbladder stones', Canadian Journal of Gastroenterology, 11 57-64 (1997)

Increasing evidence implicates prolonged intestinal transit (slow transit constipation) in the pathogenesis of conventional gallbladder stones (GBS), and that of gallstones induce... [more]

Increasing evidence implicates prolonged intestinal transit (slow transit constipation) in the pathogenesis of conventional gallbladder stones (GBS), and that of gallstones induced by long term octreotide (OT) treatment. Both groups of GBS patients have multiple abnormalities in the lipid composition and physical chemistry of their gallbladder bile - associated with, and possibly due to, an increased proportion of deoxycholic acid (DCA) (percentage of total bile acids). In turn, this increase in the percentage of DCA seems to be a consequence of prolonged colonic transit. Thus, in acromegalic patients OT treatment significantly prolongs large bowel transit time (LBTT) and leads to an associated increase of the percentage of DCA in fasting serum (and, by implication, in gallbladder bile). LBTT is linearly related to the percentage of DCA in fasting serum and correlates significantly with DCA input (into the enterohepatic circulation) and DCA pool size. However, these advances effects of OT can be overcome by the concomitant use of the prokinetic drug cisapride, which normalizes LBTT and prevents the rise in the percentage of serum DCA. Therefore, in OT-treated patients and other groups at high risk of developing stones, it may be possible to prevent GBS formation with the use of intestinal prokinetic drugs.

Citations Scopus - 28
1997 Jenkins PJ, Crockett L, Veysey MJ, Fairclough PD, Besser GM, 'Increased serum deoxycholic acid levels in acromegalic patients with colorectal neoplasia', EUROPEAN JOURNAL OF CANCER, 33 767-767 (1997)
1996 Hussaini SH, Pereira SP, Veysey MJ, Kennedy C, Jenkins P, Murphy GM, et al., 'Roles of gall bladder emptying and intestinal transit in the pathogenesis of octreotide induced gall bladder stones', GUT, 38 775-783 (1996)
DOI 10.1136/gut.38.5.775
Citations Scopus - 51Web of Science - 54
Show 68 more journal articles

Conference (31 outputs)

Year Citation Altmetrics Link
2014 Veysey M, Siow W, Niblett S, King K, Yates Z, Lucock M, 'Hepatic fibrosis in an elderly population', JOURNAL OF GASTROENTEROLOGY AND HEPATOLOGY (2014) [E3]
Co-authors Katrina King, Mark Lucock, Zoe Yates
2014 Veysey M, Siow W, Niblett S, King K, Yates Z, Lucock M, 'White cell counts and non-alcoholic fatty liver disease', JOURNAL OF GASTROENTEROLOGY AND HEPATOLOGY (2014) [E3]
Co-authors Mark Lucock, Zoe Yates
2013 Veysey M, Siow W, Choi J-H, Martin C, Tang S, Yates ZR, Lucock M, 'A Bitter Taste Gene (P49a Variant of Tas2r38) Interacts With A1298c-MTHFR to Modify Risk for Adenomas in an Australian Population', GASTROENTEROLOGY, Orlando, FL (2013) [E3]
Co-authors Mark Lucock, Zoe Yates
2013 Choi J, Siow W, Yates Z, Lucock M, Veysey M, 'Influence of synthetic folic acid concentration on Caco-2 cell growth', JOURNAL OF GASTROENTEROLOGY AND HEPATOLOGY (2013) [E3]
Co-authors Mark Lucock, Zoe Yates
2013 Siow W, Niblett S, King K, Yates Z, Hampe T, Lucock M, Veysey M, 'Prevalence of non-alcoholic fatty liver disease in an elderly Australian population', JOURNAL OF GASTROENTEROLOGY AND HEPATOLOGY (2013) [E3]
Co-authors Katrina King, Zoe Yates, Mark Lucock
2013 Siow W, Niblett S, King K, Yates Z, Martin C, Lucock M, Veysey M, 'A community-based study of dietary macro and micronutrients and the risk of colorectal cancer in an elderly Australian population', JOURNAL OF GASTROENTEROLOGY AND HEPATOLOGY (2013) [E3]
Co-authors Zoe Yates, Mark Lucock, Katrina King
2012 Kostalas S, Veysey M, 'IF YOU DON'T TAKE A TEMPERATURE YOU CAN'T FIND A FEVER', INTERNAL MEDICINE JOURNAL (2012) [E3]
2011 Choi J-H, Yates ZR, Boyd L, Veysey MJ, Lucock MD, 'Dietary folate vitamers as potential risk factors in the aetiology of adenomatous polyps', Australasian Medical Journal, Queenstown, NZ (2011) [E3]
Co-authors Mark Lucock, Zoe Yates
2011 Yates ZR, Kho J, Choi J-H, Boyd L, Ng X, Skinner V, et al., 'C776G TCNII genotype influences the relationship between blood vitamin B12 and cellular folate', Australasian Medical Journal, Queenstown, NZ (2011) [E3]
Co-authors Mark Lucock, Zoe Yates
2011 Naylor C, Lucock MD, Veysey MJ, Naumovski N, Boyd L, Dufficy L, et al., 'Folate nutritional genetics and degenerative disorders in the elderly with special reference to hypertension and depression', Australasian Medical Journal, Queenstown, NZ (2011) [E3]
Co-authors Mark Lucock, Zoe Yates, Paul Roach
2011 Kostalas S, Dowsett J, Gilbert D, Gill R, Panetta J, Singh S, et al., 'Terlipressin is associated hyponatremia', JOURNAL OF GASTROENTEROLOGY AND HEPATOLOGY (2011) [E3]
2010 Chang J, Lucock MD, Wai R, Boyd L, Ng X, Skinner VM, et al., 'Dietary folic acid, red cell folate and the risk of adenomatous polyps in an Australian population', Journal of Gastroenterology and Hepatology. Special Issue: Australian Gastroenterology Week 2010, Gold Coast, QLD (2010) [E3]
Co-authors Zoe Yates, Paul Roach, Mark Lucock
2009 Veysey MJ, Boyd L, Wai R, Ng X, Skinner V, Tang S, et al., 'Preliminary data to support a relationship between taste genetics, folate status, folate genes and the development of colonic adenomas: A novel nutrigenomic circuit', Gastro 2009 UEGW/WCOG: Scientific Programme & EACCME, London, UK (2009) [E3]
Co-authors Zoe Yates, Mark Lucock, Paul Roach
2009 Boyd L, Lucock MD, Wai R, Ng X, Yates ZR, Skinner V, et al., 'Folate status: A recognised determinant of colorectal neoplasia may be modified by bitter taste perception and genetics', Proceedings of the Nutrition Society of Australia, Newcastle, NSW (2009) [E3]
Co-authors Paul Roach, Zoe Yates, Mark Lucock
2008 Ng X, Boyd L, Dufficy L, Naumovski N, Blades BL, Travers C, et al., 'Folate genes and risk for hypertension in an elderly population sample', 3rd Asia Pacific Nutrigenomics Conference: Conference Program & Information, Melbourne, VIC (2008) [E3]
Co-authors Zoe Yates, Mark Lucock, Paul Roach
2008 Blacklaws H, Veysey H, Skinner V, Sheather-Reid R, Hawken G, Veysey M, 'Interferon and ribavirin combination therapy for chronic hepatitis C: a family impact study', JOURNAL OF GASTROENTEROLOGY AND HEPATOLOGY (2008) [E3]
2008 Wai RKH, Boyd L, Ng X, King K, Skinner V, Roach PD, et al., 'Preliminary evidence that bitter taste perception can modify folate status: A recognised risk factor for colorectal neoplasia', Journal of Gastroenterology and Hepatology, Brisbane, QLD (2008) [E3]
Co-authors Zoe Yates, Paul Roach, Mark Lucock
2008 Boyd L, Wai RKH, Ng X, King K, Skinner V, Roach PD, et al., 'A preliminary study to examine whether common folate polymorphisms are risk factors for the development of adenomatous polyps', Journal of Gastroenterology and Hepatology, Brisbane, QLD (2008) [E3]
Co-authors Zoe Yates, Paul Roach, Mark Lucock
2006 Roach PD, Dufficy L, Naumovski N, Ng X, Blades BL, Travers C, et al., 'The association of dietary folate with serum and red cell folate is modulated by the G80A reduced folate carrier single nucloetide polymorphism in an elderly population sample', The Proceedings of the Nutrition Society of Australia, Sydney, NSW (2006) [E3]
Co-authors Mark Lucock, Paul Roach
2006 Veysey M, King K, Walsh P, Vigenser B, Leijten W, 'Head-to-head comparison of the national bowel cancer screening pilot and rotary bowelscan', JOURNAL OF GASTROENTEROLOGY AND HEPATOLOGY (2006)
2006 Blades BL, Naumovski N, Roach PD, Lucock MD, Travers C, Lewis PR, et al., 'Vitamin B12 is a better inverse correlate for homocysteine than folate in an elderly population sample', Atherosclerosis Supplements, Rome, Italy (2006) [E3]
Co-authors Mark Lucock, Paul Roach
2006 Roach PD, Naumovski N, Blades BL, Lucock MD, Travers C, Lewis PR, et al., 'Serum vitamin B12 is inversely related to blood pressure and anxiety in elderly women but not in elderly men', Atherosclerosis Supplements, Rome, Italy (2006) [E3]
Co-authors Paul Roach, Mark Lucock
2006 Naumovski N, Blades BL, Lucock MD, Travers C, Lewis PR, Sturm J, Veysey MJ, 'Serum vitamin B12 is inversely related to blood pressure and anxiety in women but not men in an elderly population sample', Heart Foundation Conference and Scientific Meeting, Sydney, NSW, Australia (2006) [E3]
Co-authors Mark Lucock
2006 Naumovski N, Roach PD, Blades BL, Lucock MD, Travers C, Lewis PR, et al., 'Homocysteine, vitamin B12 and folate in an elderly population sample', Heart Foundation Conference and Scientific Meeting, Sydney, NSW, Australia (2006) [E3]
Co-authors Paul Roach, Mark Lucock
2006 Roach PD, Dufficy L, Naumovski N, Ng X, Blades BL, Travers C, et al., 'The association of blood pressure with dietary folate in elderly females and its modulation by the G80A reduced folate carrier SNP', Program & Abstracts Australian Atherosclerosis Society, Couran Cove Island, Queensland (2006) [E3]
Co-authors Paul Roach, Mark Lucock
2005 Roach PD, Naumovski N, Dufficy L, Lucock MD, Blades BL, Lewis (Ext) PR, et al., 'Folate, viamin B12, plasma thiols and cognitive funciton in an elderly population sample', Asia Pacific Journal of Clinical Nutrition, Melbourne, Victoria (2005) [E3]
Co-authors Paul Roach, Mark Lucock
2005 Roach PD, Blades BL, Lucock MD, Naumovski N, Dufficy L, Lewis (Ext) PR, et al., 'Serum Vitamin B12 is Inversely Related to Blood Pressure in Women but not Men in an Elderly Population Sample', Proceedings of Annual Scientific Meeting 2005, Darwin, NT (2005) [E3]
Co-authors Mark Lucock, Paul Roach
2000 Dowling RH, Hussaini SH, Veysey MJ, Thomas LA, French GL, Wass JAH, Murphy GM, 'The octreotide model', BILE ACIDS IN HEPATOBILIARY DISEASE, ROYAL INST GREAT BRITAIN, LONDON, ENGLAND (2000)
2000 Stellini M, Veysey MJ, Thomas LA, Milovic V, Jenkins PJ, Fairclough P, et al., 'Role of bile acids in colonic neoplasia - human studies', BILE ACIDS IN HEPATOBILIARY DISEASE, ROYAL INST GREAT BRITAIN, LONDON, ENGLAND (2000)
Citations Web of Science - 1
1999 Thomas LA, Bathgate T, Veysey MJ, Smeeton N, French GL, Murphy GM, Dowling RH, 'Transit-induced changes in colonic bacteriology, bile acid metabolizing enzymes and luminal pH influence deoxycholic acid metabolism', BILE ACIDS AND CHOLESTASIS, TITISEE, GERMANY (1999)
Citations Web of Science - 2
1997 Veysey MJ, Arraton SRD, Mallet A, Jenkins P, Murphy GM, Wass JAH, Dowling RH, 'Does cisapride overcome the effects of octreotide on intestinal transit, thereby reducing the proportion of deoxycholic acid in bile and serum?', BILE ACIDS IN HEPATOBILIARY DISEASES: BASIC RESEARCH AND CLINICAL APPLICATION, FREIBURG, GERMANY (1997)
Citations Web of Science - 1
Show 28 more conferences
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Grants and Funding

Summary

Number of grants 13
Total funding $1,313,736

Click on a grant title below to expand the full details for that specific grant.


20152 grants / $421,000

Is retirement village living good for health: Comparing the health of older adults living in retirement villages and the community$341,000

Funding body: Central Coast Local Health District

Funding body Central Coast Local Health District
Project Team Associate Professor Martin Veysey, Doctor Katrina King, Doctor Suzanne Niblett
Scheme Research Grant
Role Lead
Funding Start 2015
Funding Finish 2015
GNo G1401449
Type Of Funding Other Public Sector - State
Category 2OPS
UON Y

"Same same but different" - how different institutional settings impact the delivery of a joint medical curriculum$80,000

Funding body: Central Coast Local Health District

Funding body Central Coast Local Health District
Project Team Associate Professor Martin Veysey, Doctor Robbert Duvivier, Doctor Caragh Brosnan
Scheme Research Sponsorship Scholarship
Role Lead
Funding Start 2015
Funding Finish 2015
GNo G1500446
Type Of Funding Other Public Sector - State
Category 2OPS
UON Y

20141 grants / $25,000

Activating natural protective and healing responses in chronic inflammatory bowel disease$25,000

Funding body: Hunter Medical Research Institute

Funding body Hunter Medical Research Institute
Project Team Doctor Simon Keely, Doctor Ellen Marks, Associate Professor Martin Veysey
Scheme Research Grant
Role Investigator
Funding Start 2014
Funding Finish 2014
GNo G1401453
Type Of Funding Grant - Aust Non Government
Category 3AFG
UON Y

20131 grants / $65,624

Development of a clinical supervision professional development program for registrars & Career Medical Officers (CMOs) across Hunter & Coast ICTN$65,624

Funding body: HETI (Health Education and Training Institute)

Funding body HETI (Health Education and Training Institute)
Project Team Professor Brian Jolly, Conjoint Associate Professor Jane Conway, Professor Kichu Nair, Associate Professor Martin Veysey
Scheme NSW ICTN Local Project Fund
Role Investigator
Funding Start 2013
Funding Finish 2013
GNo G1201154
Type Of Funding Other Public Sector - State
Category 2OPS
UON Y

20121 grants / $24,500

Phase I and II detoxification genes; the correlation of epigenetic modifications with nutritional status, lifestyle factors, health and disease$24,500

Funding body: CSIRO - Energy Technology

Funding body CSIRO - Energy Technology
Project Team Associate Professor Mark Lucock, Associate Professor Martin Veysey, Doctor Zoe Yates, Miss Emma Beckett, Dr Konsta Duesing
Scheme Postgraduate Research Scholarship
Role Investigator
Funding Start 2012
Funding Finish 2012
GNo G1200845
Type Of Funding Other Public Sector - Commonwealth
Category 2OPC
UON Y

20101 grants / $36,364

Is retirement village living good for health: Comparing the health of older adults living in retirement villages and the community$36,364

Funding body: Valhalla Village Pty Ltd

Funding body Valhalla Village Pty Ltd
Project Team Associate Professor Martin Veysey, Dr Peter Lewis, Associate Professor Mark Lucock, Doctor Paul Roach, Dr David Kennedy
Scheme Linkage Projects Partner funding
Role Lead
Funding Start 2010
Funding Finish 2010
GNo G1000936
Type Of Funding Aust Competitive - Commonwealth
Category 1CS
UON Y

20091 grants / $61,281

The Association between Folate Nutritional Status and Folate Gene Polymorphisms in an Elderly Australian Population$61,281

Funding body: Central Coast Local Health District

Funding body Central Coast Local Health District
Project Team Associate Professor Martin Veysey, Doctor Zoe Yates, Associate Professor Mark Lucock, Doctor Paul Roach
Scheme Research Sponsorship Scholarship
Role Lead
Funding Start 2009
Funding Finish 2009
GNo G0190658
Type Of Funding Other Public Sector - State
Category 2OPS
UON Y

20084 grants / $660,923

Is retirement village living good for health: Comparing the health of older adults living in retirement villages and the community$390,173

Funding body: ARC (Australian Research Council)

Funding body ARC (Australian Research Council)
Project Team Associate Professor Martin Veysey, Dr Peter Lewis, Associate Professor Mark Lucock, Doctor Paul Roach, Dr David Kennedy
Scheme Linkage Projects
Role Lead
Funding Start 2008
Funding Finish 2008
GNo G0188386
Type Of Funding Aust Competitive - Commonwealth
Category 1CS
UON Y

Is retirement village living good for health: Comparing the health of older adults living in retirement villages and the community$170,750

Funding body: Central Coast Local Health District

Funding body Central Coast Local Health District
Project Team Associate Professor Martin Veysey, Dr Peter Lewis, Associate Professor Mark Lucock, Doctor Paul Roach, Dr David Kennedy
Scheme Linkage Projects Partner funding
Role Lead
Funding Start 2008
Funding Finish 2008
GNo G0188995
Type Of Funding Aust Competitive - Commonwealth
Category 1CS
UON Y

Is retirement village living good for health: Comparing the health of older adults living in retirement villages and the community$85,000

Funding body: UnitingCare Ageing NSW.ACT

Funding body UnitingCare Ageing NSW.ACT
Project Team Associate Professor Martin Veysey, Dr Peter Lewis, Associate Professor Mark Lucock, Doctor Paul Roach, Dr David Kennedy
Scheme Linkage Projects Partner funding
Role Lead
Funding Start 2008
Funding Finish 2008
GNo G0189230
Type Of Funding Aust Competitive - Commonwealth
Category 1CS
UON Y

Is retirement village living good for health: Comparing the health of older adults living in retirement villages and the community$15,000

Funding body: Urbis Pty Ltd

Funding body Urbis Pty Ltd
Project Team Associate Professor Martin Veysey, Dr Peter Lewis, Associate Professor Mark Lucock, Doctor Paul Roach, Dr David Kennedy
Scheme Linkage Projects Partner funding
Role Lead
Funding Start 2008
Funding Finish 2008
GNo G0189232
Type Of Funding Aust Competitive - Commonwealth
Category 1CS
UON Y

20051 grants / $9,044

Central Coast Vascular Health in Retirement Village Residents Study, Phase 1$9,044

Funding body: University of Newcastle

Funding body University of Newcastle
Project Team Associate Professor Martin Veysey, Associate Professor Mark Lucock
Scheme Project Grant
Role Lead
Funding Start 2005
Funding Finish 2005
GNo G0184680
Type Of Funding Internal
Category INTE
UON Y

20041 grants / $10,000

B-vitamin nutrigenomics in risk for, and progression of, dementia$10,000

Funding body: University of Newcastle

Funding body University of Newcastle
Project Team Associate Professor Martin Veysey
Scheme New Staff Grant
Role Lead
Funding Start 2004
Funding Finish 2004
GNo G0183895
Type Of Funding Internal
Category INTE
UON Y
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Research Supervision

Number of supervisions

Completed3
Current3

Total current UON EFTSL

PhD0.55

Current Supervision

Commenced Level of Study Research Title / Program / Supervisor Type
2014 PhD Vitamin D Status of Older People in Relation to Non-Communicable Disease
Medical Science, Faculty of Health and Medicine, The University of Newcastle
Co-Supervisor
2012 PhD The Association of MIRNA Profiles and DNA Methylation with Micronutrient Intake & Status
Food Science & Biotechnology, Faculty of Science and Information Technology, The University of Newcastle
Co-Supervisor
2010 PhD Genotype-Phenotype Relationships Relevant to the Lifecycle with Special Reference to Vitamin Nutrition
Food Science & Biotechnology, Faculty of Science and Information Technology, The University of Newcastle
Co-Supervisor

Past Supervision

Year Level of Study Research Title / Program / Supervisor Type
2014 PhD The Role of Folic Acid Related Nutritional Genetics in Common Chronic Degenerative Disorders
Food Science & Biotechnology, Faculty of Science and Information Technology, The University of Newcastle
Co-Supervisor
2014 PhD In Vitro and In Vivo Approaches to the Examination of Folate-Related Nutritional Genetics in Health and Disease
Food Science & Biotechnology, Faculty of Science and Information Technology, The University of Newcastle
Co-Supervisor
2009 PhD Physico-Chemical Properties and Genetic Factors That Influence the Bioavailability and Metabolism of Folic Acid: Implications in Health and Diseases
Food Science & Biotechnology, Faculty of Science and Information Technology, The University of Newcastle
Co-Supervisor
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Associate Professor Martin Veysey

Position

Associate Professor
Central Coast Health Teaching and Research
School of Medicine and Public Health
Faculty of Health and Medicine

Contact Details

Email martin.veysey@newcastle.edu.au
Phone (02) 4320 3022
Fax (02) 4320 3508

Office

Room GOS
Building Gosford Hospital
Location Gosford
Cnr Henry Parry Drive and Margin Street
Gosford, NSW 2250
Australia
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