Dr Grace Burns

Dr Grace Burns

Postdoctoral Researcher

School of Biomedical Sciences and Pharmacy

Career Summary

Biography

Dr. Grace Burns is a postdoctoral researcher in Immunology and Microbiology, working in the Centre of Research Excellence in Digestive Health. Her current research examines the influence of circadian rhythm on immune and microbiome-related symptoms in gastrointestinal disease. In addition, she is continuing her work on how food and microbial antigens are associated with the immune response in gastrointestinal conditions and the contribution of gut homeostatic imbalance in the small intestine to inappropriate responses to food antigens.

In 2021, Grace was awarded her PhD, which focused on elucidating immune pathways that drive the onset and continuation of symptoms in FD. The lack of knowledge surrounding the pathophysiological features and immune activation of FD has led to the diagnosis of this condition being solely reliant on self-reported symptoms and a negative endoscopic investigation. Consequently, patient management is complicated by a lack of treatable pathology or effective therapeutic options. Grace’s work in this area is focused on demonstrating that functional gastrointestinal conditions are driven by disruptions in homeostasis, activating the immune system and subsequently driving symptom onset. Her work has identified a link between a specific alteration in the small intestinal microbiota and activation of the immune system in these patients, representing a potential diagnostic test to replace the current symptom-based diagnostic process for FD. In addition, her work aimed to characterise the immune cell populations that are altered between FD patients and a control cohort, with the aim of distinguishing specific mechanisms driving disease.

Grace was awarded a Bachelor of Medical Science (Pathology)/Bachelor of Forensic Biotechnology double degree in 2015 from Charles Sturt University, Wagga Wagga. After moving to Newcastle, she completed a Bachelor of Biomedical Science (Honours First Class) qualification in 2016 under the supervision of Prof. Simon Keely and A/Prof. Jay Horvat, investigating the development of immune responses to food proteins following antibiotic treatment.


Qualifications

  • Doctor of Philosophy, University of Newcastle

Keywords

  • Circadian rhythms
  • Functional dyspepsia
  • Functional gastrointestinal disorders
  • Gastrointestinal disease
  • Gastrointestinal immunology
  • Immunology
  • Irritable bowel syndrome

Languages

  • English (Mother)

Fields of Research

Code Description Percentage
320209 Gastroenterology and hepatology 40
320404 Cellular immunology 40
320499 Immunology not elsewhere classified 20

Professional Experience

UON Appointment

Title Organisation / Department
Postdoctoral Researcher University of Newcastle
School of Biomedical Sciences and Pharmacy
Australia

Professional appointment

Dates Title Organisation / Department
1/9/2020 - 26/2/2021 Research Assistant College of Health, Medicine and Wellbeing, University of Newcastle
Australia

Awards

Award

Year Award
2019 Travel Grant - United European Gastroenterology Week
United European Gastroenterology Week
2019 Priority Research Centre for Digestive Health & Neurogastroenterology Travel Grant
The Priority Research Centre for Digestive Health & Neurogastroenterology, The University of Newcastle
2019 Tubingen Australia Psycho-Neuro-Gastroenterology Summer School Travel Grant
DAAD, German Academic Exchange Service
2018 Priority Research Centre for Digestive Health & Neurogastroenterology Travel Grant
The Priority Research Centre for Digestive Health & Neurogastroenterology, The University of Newcastle

Prize

Year Award
2018 Poster of Distinction, Experimental Biology Conference, San Diego, USA
American Physiological Society

Scholarship

Year Award
2016 Priority Research Centre for Digestive Health & Neurogastroenterology Honours Scholarship
The Priority Research Centre for Digestive Health & Neurogastroenterology, The University of Newcastle
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Publications

For publications that are currently unpublished or in-press, details are shown in italics.


Journal article (21 outputs)

Year Citation Altmetrics Link
2021 Hoedt EC, Shanahan ER, Keely S, Shah A, Burns GL, Holtmann GJ, et al., 'Draft Genome Sequence of Streptococcus salivarius AGIRA0003, Isolated from Functional Gastrointestinal Disorder Duodenal Tissue.', Microbiol Resour Announc, 10 e0075821 (2021)
DOI 10.1128/MRA.00758-21
Co-authors Emily Hoedt, Simon Keely, Nicholas Talley
2021 Burns GL, Hoedt EC, Walker MM, Talley NJ, Keely S, 'Physiological mechanisms of unexplained (functional) gastrointestinal disorders.', J Physiol, (2021)
DOI 10.1113/JP281620
Co-authors Marjorie Walker, Nicholas Talley, Simon Keely, Emily Hoedt
2021 Duncanson K, Burns G, Pryor J, Keely S, Talley NJ, 'Mechanisms of Food-Induced Symptom Induction and Dietary Management in Functional Dyspepsia.', Nutrients, 13 (2021)
DOI 10.3390/nu13041109
Citations Scopus - 5Web of Science - 2
Co-authors Nicholas Talley, Kerith Duncanson, Simon Keely
2021 Talley NJ, Walker MM, Jones M, Keely S, Koloski N, Cameron R, et al., 'Letter: budesonide for functional dyspepsia with duodenal eosinophilia-randomised, double-blind, placebo-controlled parallel-group trial.', Aliment Pharmacol Ther, 53 1332-1333 (2021)
DOI 10.1111/apt.16396
Citations Scopus - 1Web of Science - 1
Co-authors Marjorie Walker, Simon Keely, Nicholas Talley
2021 Burns GL, Hoedt EC, Keely S, 'Spore-forming probiotics for functional dyspepsia.', Lancet Gastroenterol Hepatol, 6 772-773 (2021)
DOI 10.1016/S2468-1253(21)00260-0
Co-authors Simon Keely, Emily Hoedt
2021 Burns GL, Bruce JK, Cameron R, Potter MD, Minahan K, Mathe A, et al., '463 ALLERGIC-LIKE EFFECTOR MEMORY T HELPER (TH) 2 AND AUTOIMMUNE-LIKE TH17.1 CELL POPULATIONS ARE INCREASED IN THE DUODENUM OF PATIENTS WITH FUNCTIONAL DYSPEPSIA', Gastroenterology, 160 S-95 (2021)
DOI 10.1016/s0016-5085(21)00968-9
2021 Burns GL, Potter MD, Mathe A, Bruce JK, Minahan K, Barnes J, et al., 'Fr237 MUCOSAL EFFECTOR T HELPER 17 RESPONSES TO GLUTEN STIMULATION ARE ASSOCIATED WITH GENE EXPRESSION OF TRAV26-2, A GLIADIN-BIASED T CELL RECEPTOR VARIANT IN PATIENTS WITH FUNCTIONAL DYSPEPSIA.', Gastroenterology, 160 S-273 (2021)
DOI 10.1016/s0016-5085(21)01380-9
2020 Carroll GM, Burns GL, Petit JA, Walker MM, Mathe A, Smith SR, et al., 'Does postoperative inflammation or sepsis generate neutrophil extracellular traps that influence colorectal cancer progression? A systematic review', Surgery Open Science, 2 57-69 (2020) [C1]
DOI 10.1016/j.sopen.2019.12.005
Co-authors Andrea Johns, Marjorie Walker, Stephen Smith, Peter Pockney, Simon Keely
2020 Talley NJ, Holtmann GJ, Jones M, Koloski NA, Walker MM, Burns G, et al., 'Zonulin in serum as a biomarker fails to identify the IBS, functional dyspepsia and non-coeliac wheat sensitivity', GUT, 69 1719-1722 (2020)
DOI 10.1136/gutjnl-2019-318664
Citations Scopus - 7Web of Science - 7
Co-authors Marjorie Walker, Nicholas Talley, Simon Keely
2020 Wauters L, Burns G, Ceulemans M, Walker MM, Vanuytsel T, Keely S, Talley NJ, 'Duodenal inflammation: an emerging target for functional dyspepsia?', Expert Opinion on Therapeutic Targets, 24 511-523 (2020) [C1]

Introduction: Functional dyspepsia (FD) is one of the most common functional gastrointestinal disorders and is classified into postprandial distress and epigastric pain syndrome. ... [more]

Introduction: Functional dyspepsia (FD) is one of the most common functional gastrointestinal disorders and is classified into postprandial distress and epigastric pain syndrome. Despite the recognition of duodenal inflammation as a potential trigger of symptoms, only limited anti-inflammatory therapies exist. Areas covered: This narrative review summarizes the recent advances in the pathophysiology and treatment of FD; it identifies potential therapeutic targets and gaps in the field. An electronic literature search was conducted in Pubmed up to 31st of December 2019. Expert opinion: There is compelling evidence for the role of duodenal inflammation and the eosinophil-mast cell axis in the pathogenesis of dyspeptic symptoms. Traditional prokinetic drugs and neuromodulators target gastric dysmotility and visceral hypersensitivity but are hampered by limited efficacy and side effects. Independent of acid suppression, the anti-inflammatory action of proton pump inhibitors, which remain the first-line therapy in FD, may also explain their therapeutic effect. Other existing and newly established anti-inflammatory drugs should be investigated while trials including probiotics and selective antibiotics should examine the host microbiome and immune activation. Targeted treatments for potential causes of duodenal pathology, such as impaired permeability and dysbiosis, are likely to emerge in the future.

DOI 10.1080/14728222.2020.1752181
Citations Scopus - 9Web of Science - 9
Co-authors Nicholas Talley, Simon Keely, Marjorie Walker
2020 Pryor J, Burns GL, Duncanson K, Horvat JC, Walker MM, Talley NJ, Keely S, 'Functional Dyspepsia and Food: Immune Overlap with Food Sensitivity Disorders.', Current gastroenterology reports, 22 (2020) [C1]
DOI 10.1007/s11894-020-00789-9
Citations Scopus - 5
Co-authors Kerith Duncanson, Jay Horvat, Simon Keely, Nicholas Talley, Marjorie Walker
2019 B Biomed GB, Carroll G, Mathe A, Horvat J, Foster P, Walker MM, et al., 'Evidence for Local and Systemic Immune Activation in Functional Dyspepsia and the Irritable Bowel Syndrome: A Systematic Review', American Journal of Gastroenterology, 114 429-436 (2019) [C1]

BACKGROUND:Subtle histopathologic features such as eosinophilia and increased mast cells have been observed in functional gastrointestinal disorders (FGIDs), including functional ... [more]

BACKGROUND:Subtle histopathologic features such as eosinophilia and increased mast cells have been observed in functional gastrointestinal disorders (FGIDs), including functional dyspepsia (FD) and the irritable bowel syndrome (IBS). The mechanisms that drive recruitment of these cells to the gastrointestinal tract remain unexplained, largely due to the heterogeneity in phenotypes among patients diagnosed with such conditions. We aimed to systematically review the literature and collate the evidence for immune activation in FD and IBS, and where possible, detail the nature of activation.METHODS:Seven literature databases were searched using the keywords: 'functional gastrointestinal disorder', FGID, 'functional dyspepsia', 'non-ulcer dyspepsia', 'idiopathic dyspepsia', 'irritable bowel syndrome', IBS and 'immun*'.RESULTS:Fifty-one papers reporting discordant immune features met the selection criteria for this review. Changes in lymphocyte populations, including B and T lymphocyte numbers and activation status were reported in IBS and FD, in conjunction with duodenal eosinophilia in FD and increased colonic mast cells in IBS. Increases in circulating a4+ß7+ gut-homing T cells appear to be linked to the pathophysiology of both FD and IBS. Studies in the area are complicated by poor phenotyping of patients into subgroups and the subtle nature of the immune activity involved in FD and IBS.CONCLUSIONS:Alterations in proportions of gut-homing T lymphocytes in both FD and IBS indicate that a loss of mucosal homeostasis may drive the symptoms of FD and IBS. There is indirect evidence that Th17 responses may play a role in FGIDs, however the evidence for a Th2 immune phenotype in FD and IBS is limited. Although immune involvement is evident, large, well-characterised patient cohorts are required to elucidate the immune mechanisms driving the development of FGIDs.

DOI 10.1038/s41395-018-0377-0
Citations Scopus - 46Web of Science - 40
Co-authors Nicholas Talley, Paul Foster, Andrea Johns, Marjorie Walker, Simon Keely, Jay Horvat
2019 Burns G, Pryor J, Holtmann G, Walker MM, Talley NJ, Keely S, 'Immune Activation in Functional Gastrointestinal Disorders.', Gastroenterology & hepatology, 15 539-548 (2019) [C1]
Citations Scopus - 7
Co-authors Nicholas Talley, Simon Keely, Marjorie Walker
2019 Burns G, Carroll G, Mathe A, Horvat J, Foster P, Walker MM, et al., 'Evidence for Local and Systemic Immune Activation in Functional Dyspepsia and the Irritable Bowel Syndrome: A Systematic Review.', Am J Gastroenterol, 114 429-436 (2019)
DOI 10.1038/s41395-018-0377-0
Co-authors Simon Keely
2019 Talley NJ, Holtmann G, Walker MM, Burns G, Potter M, Shah A, et al., 'Circulating Anti-cytolethal Distending Toxin B and Anti-vinculin Antibodies as Biomarkers in Community and Healthcare Populations With Functional Dyspepsia and Irritable Bowel Syndrome.', Clin Transl Gastroenterol, 10 e00064 (2019) [C1]
DOI 10.14309/ctg.0000000000000064
Citations Scopus - 13Web of Science - 13
Co-authors Simon Keely, Nicholas Talley, Marjorie Walker
2019 Liu G, Mateer SW, Hsu A, Goggins BJ, Tay H, Mathe A, et al., 'Platelet activating factor receptor regulates colitis-induced pulmonary inflammation through the NLRP3 inflammasome', Mucosal Immunology, 12 862-873 (2019) [C1]

Extra-intestinal manifestations (EIM) are common in inflammatory bowel disease (IBD). One such EIM is sub-clinical pulmonary inflammation, which occurs in up to 50% of IBD patient... [more]

Extra-intestinal manifestations (EIM) are common in inflammatory bowel disease (IBD). One such EIM is sub-clinical pulmonary inflammation, which occurs in up to 50% of IBD patients. In animal models of colitis, pulmonary inflammation is driven by neutrophilic infiltrations, primarily in response to the systemic bacteraemia and increased bacterial load in the lungs. Platelet activating factor receptor (PAFR) plays a critical role in regulating pulmonary responses to infection in conditions, such as chronic obstructive pulmonary disease and asthma. We investigated the role of PAFR in pulmonary EIMs of IBD, using dextran sulfate sodium (DSS) and anti-CD40 murine models of colitis. Both models induced neutrophilic inflammation, with increased TNF and IL-1ß levels, bacterial load and PAFR protein expression in mouse lungs. Antagonism of PAFR decreased lung neutrophilia, TNF, and IL-1ß in an NLRP3 inflammasome-dependent manner. Lipopolysaccharide from phosphorylcholine (ChoP)-positive bacteria induced NLRP3 and caspase-1 proteins in human alveolar epithelial cells, however antagonism of PAFR prevented NLRP3 activation by ChoP. Amoxicillin reduced bacterial populations in the lungs and reduced NLRP3 inflammasome protein levels, but did not reduce PAFR. These data suggest a role for PAFR in microbial pattern recognition and NLRP3 inflammasome signaling in the lung.

DOI 10.1038/s41385-019-0163-3
Citations Scopus - 28Web of Science - 27
Co-authors Philip Hansbro, Simon Keely, Alan Hsu, Andrea Johns, Peter Wark, Bridie Goggins, Michael Fricker, Steven Maltby, Paul Foster, Hock Tay
2018 Mateer SW, Mathe A, Bruce J, Liu G, Maltby S, Fricker M, et al., 'IL-6 Drives Neutrophil-Mediated Pulmonary Inflammation Associated with Bacteremia in Murine Models of Colitis', American Journal of Pathology, 188 1625-1639 (2018) [C1]

Inflammatory bowel disease (IBD) is associated with several immune-mediated extraintestinal manifestations. More than half of all IBD patients have some form of respiratory pathol... [more]

Inflammatory bowel disease (IBD) is associated with several immune-mediated extraintestinal manifestations. More than half of all IBD patients have some form of respiratory pathology, most commonly neutrophil-mediated diseases, such as bronchiectasis and chronic bronchitis. Using murine models of colitis, we aimed to identify the immune mechanisms driving pulmonary manifestations of IBD. We found increased neutrophil numbers in lung tissue associated with the pulmonary vasculature in both trinitrobenzenesulfonic acid¿ and dextran sulfate sodium¿induced models of colitis. Analysis of systemic inflammation identified that neutrophilia was associated with bacteremia and pyrexia in animal models of colitis. We further identified IL-6 as a systemic mediator of neutrophil recruitment from the bone marrow of dextran sulfate sodium animals. Functional inhibition of IL-6 led to reduced systemic and pulmonary neutrophilia, but it did not attenuate established colitis pathology. These data suggest that systemic bacteremia and pyrexia drive IL-6 secretion, which is a critical driver for pulmonary manifestation of IBD. Targeting IL-6 may reduce neutrophil-associated extraintestinal manifestations in IBD patients.

DOI 10.1016/j.ajpath.2018.03.016
Citations Scopus - 19Web of Science - 20
Co-authors Michael Fricker, Steven Maltby, Paul Foster, Robert Callister, Philip Hansbro, Simon Keely, Andrea Johns, Jay Horvat, Hock Tay, Bridie Goggins, Marjorie Walker
2018 Burns G, Shanahan E, Do A, Bruce J, Minahan K, Horvat J, et al., 'Seroreactivity to Microbial Antigens and Gut-Homing Immune Responses in Functional Dyspepsia Patients with Postprandial Distress Syndrome', The FASEB Journal, 32 (2018)
DOI 10.1096/fasebj.2018.32.1_supplement.613.3
2017 Marks E, Naudin C, Nolan G, Goggins BJ, Burns G, Mateer SW, et al., 'Regulation of IL-12p40 by HIF controls Th1/Th17 responses to prevent mucosal inflammation', MUCOSAL IMMUNOLOGY, 10 1224-1236 (2017) [C1]
DOI 10.1038/mi.2016.135
Citations Scopus - 14Web of Science - 14
Co-authors Simon Keely, Robert Callister, Martin Veysey, Paul Foster, Marjorie Walker, Nicholas Talley, Bridie Goggins
Carroll GM, Burns GL, Petit JA, Walker MM, Mathe A, Smith SR, et al., 'Does Surgery Generate Neutrophil Extracellular Traps that Influence Colorectal Cancer Progression? A Systematic Review', SSRN Electronic Journal,
DOI 10.2139/ssrn.3456869
Keely S, Burns GL, Fan K, Soh WS, Brown G, Walker MM, et al., 'Prevalence and Characteristics of Gastrointestinal Symptoms in SARS-CoV-2 Infection: Systematic Review and Meta-Analysis', SSRN Electronic Journal,
DOI 10.2139/ssrn.3590477
Show 18 more journal articles

Conference (7 outputs)

Year Citation Altmetrics Link
2021 Delforce S, Lumbers E, Burns G, Marsland M, Zakar T, Pringle K, 'THE ANTI-INFLAMMATORY ROLE OF ANGIOTENSIN-(1-7) IN HUMAN INTRAUTERINE TISSUES', PLACENTA (2021)
Co-authors Kirsty Pringle
2021 Eslick GD, Fan K, Nair PM, Burns GL, Keely S, Walker M, Talley NJ, 'Epidemiological and clinical factors associated with colonic spirochete (Brachyspira pilosicoli and Brachyspira aalborgi) infection: A pooled analysis', JOURNAL OF GASTROENTEROLOGY AND HEPATOLOGY (2021)
Co-authors Marjorie Walker, Nicholas Talley, Simon Keely
2021 Burns GL, Bruce JK, Cameron R, Potter MD, Minahan K, Mathe A, et al., 'ALLERGIC-LIKE EFFECTOR MEMORY T HELPER (TH) 2 AND AUTOIMMUNE-LIKE TH17.1 CELL POPULATIONS ARE INCREASED IN THE DUODENUM OF PATIENTS WITH FUNCTIONAL DYSPEPSIA', GASTROENTEROLOGY (2021)
Co-authors Paul Foster, Jay Horvat, Marjorie Walker, Simon Keely, Martin Veysey, Nicholas Talley, Andrea Johns
2021 Burns GL, Potter MD, Mathe A, Bruce JK, Minahan K, Barnes J, et al., 'MUCOSAL EFFECTOR T HELPER 17 RESPONSES TO GLUTEN STIMULATION ARE ASSOCIATED WITH GENE EXPRESSION OF TRAV26-2, A GLIADIN-BIASED T CELL RECEPTOR VARIANT IN PATIENTS WITH FUNCTIONAL DYSPEPSIA.', GASTROENTEROLOGY (2021)
Co-authors Kerith Duncanson, Jay Horvat, Paul Foster, Nicholas Talley, Simon Keely, Andrea Johns, Martin Veysey, Marjorie Walker
2019 Liu G, Mateer S, Hsu A, Goggins B, Tay H, Mathe A, et al., 'Platelet activating factor receptor regulates colitis-induced pulmonary inflammation through the NLRP3 inflammasome', EUROPEAN JOURNAL OF IMMUNOLOGY, Beijing, PEOPLES R CHINA (2019)
Co-authors Steven Maltby, Michael Fricker, Philip Hansbro, Hock Tay, Andrea Johns, Simon Keely, Alan Hsu, Paul Foster
2018 Burns G, Shanahan E, Anh D, Bruce J, Minahan K, Horvat J, et al., 'Seroreactivity to Microbial Antigens and Gut-Homing Immune Responses in Functional Dyspepsia Patients with Postprandial Distress Syndrome', FASEB JOURNAL, San Diego, CA (2018)
Co-authors Paul Foster, Nicholas Talley, Simon Keely, Jay Horvat, Marjorie Walker
2017 Marks E, Naudin C, Walker MM, Veysey M, Foster P, Talley NJ, et al., 'REGULATION OF IL-12P40 BY HIF CONTROLS TH1/TH17 RESPONSES TO PREVENT MUCOSAL INFLAMMATION', GASTROENTEROLOGY, Chicago, IL (2017)
DOI 10.1016/S0016-5085(17)32053-X
Citations Web of Science - 1
Co-authors Simon Keely, Martin Veysey, Robert Callister, Paul Foster, Marjorie Walker, Nicholas Talley, Bridie Goggins
Show 4 more conferences
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Grants and Funding

Summary

Number of grants 3
Total funding $46,220

Click on a grant title below to expand the full details for that specific grant.


20213 grants / $46,220

Investigating circadian rhythms and melatonin metabolism in patients with functional gastrointestinal disorders.$30,000

Funding body: NHMRC Centre of Research Excellence in Digestive Health

Funding body NHMRC Centre of Research Excellence in Digestive Health
Project Team

Dr. Grace Burns, Dr. Kerith Duncanson, L/Prof. Nicholas Talley, Prof. Simon Keely

Scheme 2020 Pilot Grant
Role Lead
Funding Start 2021
Funding Finish 2021
GNo
Type Of Funding Internal
Category INTE
UON N

Gut-homing T cells in the blood of early-stage Parkinson’s Disease patients$11,270

Funding body: 2021 Strategic Research Pilot Grant - College of Health, Medicine and Wellbeing, University of Newcastle

Funding body 2021 Strategic Research Pilot Grant - College of Health, Medicine and Wellbeing, University of Newcastle
Project Team

Dr. Grace Burns, Dr. Tamara Canento Sanchez, Dr. Emily Hoedt, Prof. Simon Keely, Dr. Elizabeth Pepper, L/Prof. Nicholas Talley

Scheme College of Health, Medicine and Wellbeing - 2021 Strategic Pilot Grant Scheme
Role Lead
Funding Start 2021
Funding Finish 2021
GNo
Type Of Funding Internal
Category INTE
UON N

Pilot data collection for analysis of the microbiome and pregnancy outcomes in the Newcastle 1000 study.$4,950

Funding body: Hunter Medical Research Institute

Funding body Hunter Medical Research Institute
Project Team Doctor Emily Hoedt, Doctor Grace Burns, Doctor Tegan Grace
Scheme Research Grant
Role Investigator
Funding Start 2021
Funding Finish 2021
GNo G2100165
Type Of Funding C3300 – Aust Philanthropy
Category 3300
UON Y
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Research Supervision

Number of supervisions

Completed0
Current2

Current Supervision

Commenced Level of Study Research Title Program Supervisor Type
2021 PhD Immune Screening as a Predictor of Food Triggers for Relapse in Crohn’s Disease. PhD (Immunology & Microbiol), College of Health, Medicine and Wellbeing, The University of Newcastle Co-Supervisor
2021 PhD Peri-Operative Factors Affecting Natural Killer Cell Function and Their Role in Colorectal Cancer Re-Occurrence and Metastasis PhD (Immunology & Microbiol), College of Health, Medicine and Wellbeing, The University of Newcastle Co-Supervisor
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Dr Grace Burns

Positions

Postdoctoral Researcher
Centre of Research Excellence in Digestive Health
School of Biomedical Sciences and Pharmacy
College of Health, Medicine and Wellbeing

Casual Research Assistant
Centre of Research Excellence in Digestive Health
School of Medicine and Public Health
College of Health, Medicine and Wellbeing

Contact Details

Email g.burns@newcastle.edu.au
Phone (02) 4042 0181

Office

Room Level 3 East
Building Hunter Medical Research Institute
Location HMRI

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