Dr Kaushik Maiti

Dr Kaushik Maiti

Research Officer

School of Medicine and Public Health

Career Summary

Biography

Dr. Kaushik Maiti completed his master degree (MSc) in biochemistry in 1997 from University of Kalyani, India. He began working as a junior research fellow in The ICMR-Virus Unit, Kolkata, gaining experience in ethnopharmacology and microbiology. He then joined the hormone research center at Chonnam National University, South Korea, as a Ph.D. student in 2000. Dr. Maiti gained his Ph.D. degree in molecular endocrinology in 2004. In his Ph.D., he worked on gonadotropin releasing hormone (GnRH) and its G-protein coupled receptors (GPCR). He developed an antagonist for type-2 gonadotropin releasing hormone receptor.  This antagonist can inhibit the growth of prostate cancer cells both in vitro and in vivo and thus have a high potential a therapeutic in treating prostate cancer. In 2006 he joined as a postdoctoral fellow in Ohio State University, USA. During this time, he gained experiences in estrogenic regulation of contraction of vascular smooth muscles. He has been working as Research Officer in Mother and Babies Research Centre (MBRC), the University of Newcastle, Australia from 2007 to present.  He is currently investigating ageing of the placenta which can be linked to unexplained antepartum stillbirth. He is also trying to identify the biomarkers in maternal blood to detect ailing foetuses. 

Dr. Maiti has published 25 papers in peer reviewed journals: Journal of clinical endocrinology and metabolism, Endocrinology, Molecular Endocrinology, Molecular Pharmacology, Cancer Research, Nature Communications and American Journal of Obstetrics and Gynecology and more. He has presented his work at national and international scientific conferences. Google scholar stated that his work had been cited 750 times, and he gained h-index of 18. He supervised two doctoral students. He has received several grants from NH&MRC and philanthropic sources (Haggarty foundation, John Hunter Hospital Charitable grant, and red nose foundation).


Qualifications

  • Doctor of Science, Chonnam National University - Korea
  • Bachelor of Science, University of Calcutta
  • Master of Science (Biochemistry), University of Kalyani - India

Keywords

  • G-protein coupled receptor, ageing
  • pregnancy, stillbirth, biochemistry, endocrinology

Languages

  • Bengali (Mother)
  • English (Fluent)

Fields of Research

Code Description Percentage
119999 Medical and Health Sciences not elsewhere classified 100

Professional Experience

UON Appointment

Title Organisation / Department
Research Officer University of Newcastle
School of Medicine and Public Health
Australia
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Publications

For publications that are currently unpublished or in-press, details are shown in italics.


Journal article (25 outputs)

Year Citation Altmetrics Link
2018 Sultana Z, Maiti K, Dedman L, Smith R, 'Is there a role for placental senescence in the genesis of obstetric complications and fetal growth restriction?', American Journal of Obstetrics and Gynecology, 218 S762-S773 (2018) [C1]
DOI 10.1016/j.ajog.2017.11.567
Citations Scopus - 14Web of Science - 9
Co-authors Roger Smith, Zakia Sultana
2017 Maiti K, Sultana Z, Aitken RJ, Morris J, Park F, Andrew B, et al., 'Evidence that fetal death is associated with placental aging.', American journal of obstetrics and gynecology, 217 441.e1-441.e14 (2017) [C1]
DOI 10.1016/j.ajog.2017.06.015
Citations Scopus - 14Web of Science - 14
Co-authors Zakia Sultana, John Aitken, Roger Smith
2017 Sultana Z, Maiti K, Aitken J, Morris J, Dedman L, Smith R, 'Oxidative stress, placental ageing-related pathologies and adverse pregnancy outcomes', AMERICAN JOURNAL OF REPRODUCTIVE IMMUNOLOGY, 77 (2017) [C1]
DOI 10.1111/aji.12653
Citations Scopus - 50Web of Science - 48
Co-authors Roger Smith, John Aitken, Zakia Sultana
2016 De Sousa SMC, Maiti K, Smith R, McCormack AI, 'Corticotrophin-releasing hormone (CRH) expression in the dermoid component of ovarian teratomas', Journal of the European Academy of Dermatology and Venereology, 30 867-869 (2016)
DOI 10.1111/jdv.13035
Co-authors Roger Smith
2014 Parkington HC, Stevenson J, Tonta MA, Paul J, Butler T, Maiti K, et al., 'Diminished hERG K

Human ether-a-go-go-related gene (hERG) potassium channels determine cardiac action potential and contraction duration. Human uterine contractions are underpinned by an action pot... [more]

Human ether-a-go-go-related gene (hERG) potassium channels determine cardiac action potential and contraction duration. Human uterine contractions are underpinned by an action potential that also possesses an initial spike followed by prolonged depolarization. Here we show that hERG channel proteins (a-conducting and ßinhibitory subunits) and hERG currents exist in isolated patch-clamped human myometrial cells. We show that hERG channel activity suppresses contraction amplitude and duration before labour, thereby facilitating quiescence. During established labour, expression of ß-inhibitory protein is markedly enhanced, resulting in reduced hERG activity that is associated with an increased duration of uterine action potentials and contractions. Thus, changes in hERG channel activity contribute to electrophysiological mechanisms that produce contractions during labour. We also demonstrate that this system fails in women with elevated BMI, who have enhanced hERG activity as a result of low ß-inhibitory protein expression, which likely contributes to the weak contractions and poor labour outcomes observed in many obese women necessitating caesarean delivery. © 2014 Macmillan Publishers Limited.

DOI 10.1038/ncomms5108
Citations Scopus - 20Web of Science - 18
Co-authors Jonathan Paul, Roger Smith
2014 Chai SY, Smith R, Fitter JT, Mitchell C, Pan X, Ilicic M, et al., 'Increased progesterone receptor a expression in labouring human myometrium is associated with decreased promoter occupancy by the histone demethylase JARID1A', Molecular Human Reproduction, 20 442-453 (2014) [C1]

Progesterone regulates female reproductive function predominantly through two nuclear progesterone receptors (PRs), PR-A and PR-B. During human parturition myometrial PR expressio... [more]

Progesterone regulates female reproductive function predominantly through two nuclear progesterone receptors (PRs), PR-A and PR-B. During human parturition myometrial PR expression is altered to favour PR-A, which activates pro-labour genes. We have previously identified histone H3 lysine 4 trimethylation (H3K4me3) as an activator of myometrial PR-A expression at labour. To further elucidate the mechanisms regulating PR isoform expression in the human uterus at labour, we have (i) determined the methylation profile of the cytosine-guanine dinucleotides (CpG) island in the promoter region of the PR gene and (ii) identified the histone-modifying enzymes that target the H3K4me3 mark at the PR promoters in term and preterm human myometrial tissues obtained before and after labour onset. Bisulphite sequencing showed that despite overall low levels of PR CpG island methylation, there was a significant decrease in methylated CpGs with labour in both preterm (P < 0.05) and term (P < 0.01) groups downstream of the PR-B transcription start site. This methylation change was not associated with altered PR-B expression, but may contribute to the increase in PR-A expression with labour. Chromatin immunoprecipitation revealed that the histone methyltransferase, SET and MYND domain-containing protein 3 (SMYD3), bound to the PR gene at significantly higher levels at the PR-A promoter compared with the PR-B promoter (P < 0.010), with no labour-associated changes observed. The H3K4 demethylase, Jumonji AT-rich interactive domain 1A (JARID1A), also bound to the PR-A, but not to the PR-B promoter prior to term labour, and decreased significantly at the onset of labour (P = 0.014), providing a mechanism for the previously reported increase in H3K4me3 level and PR-A expression with labour. Our studies suggest that epigenetic changes mediated by JARID1A, SMYD3 and DNA methylation may be responsible, at least in part, for the functional progesterone withdrawal that precipitates human labour. © The Author 2014. Published by Oxford University Press on behalf of the European Society of Human Reproduction and Embryology. All rights reserved.

DOI 10.1093/molehr/gau005
Citations Scopus - 15Web of Science - 15
Co-authors John Fitter, Marina Ilicic, Roger Smith
2013 Smith R, Maiti K, Aitken RJ, 'Unexplained antepartum stillbirth: A consequence of placental aging?', PLACENTA, 34 310-313 (2013) [C3]
DOI 10.1016/j.placenta.2013.01.015
Citations Scopus - 23Web of Science - 22
Co-authors Roger Smith, John Aitken
2013 Chen Y, Allars M, Pan X, Maiti K, Angeli G, Smith R, Nicholson RC, 'Effects of corticotrophin releasing hormone (CRH) on cell viability and differentiation in the human BeWo choriocarcinoma cell line: a potential syncytialisation inducer distinct from cyclic adenosine monophosphate (cAMP)', REPRODUCTIVE BIOLOGY AND ENDOCRINOLOGY, 11 (2013) [C1]
DOI 10.1186/1477-7827-11-30
Citations Scopus - 4Web of Science - 3
Co-authors Roger Smith, Giavanna Angeli
2012 Smith R, Maiti K, 'The placenta, a transducer linking maternal nutrition to adult disease in the offspring?', Endocrinology, 153 1572-1574 (2012) [C3]
Citations Scopus - 2Web of Science - 2
Co-authors Roger Smith
2012 Tolosa Gonzalez JM, Schjenken JE, Clifton VL, Vargas A, Barbeau B, Lowry P, et al., 'The endogenous retroviral envelope protein syncytin-1 inhibits LPS/PHA-stimulated cytokine responses in human blood and is sorted into placental exosomes', Placenta, 33 933-941 (2012) [C1]
Citations Scopus - 59Web of Science - 54
Co-authors Roger Smith
2012 Smith R, Paul JW, Maiti K, Tolosa Gonzalez JM, Madsen GM, 'Recent advances in understanding the endocrinology of human birth', Trends in Endocrinology & Metabolism, 23 516-523 (2012) [C1]
Citations Scopus - 14Web of Science - 12
Co-authors Roger Smith, Jonathan Paul
2011 Paul JW, Maiti K, Read MA, Hure AJ, Smith JI, Chan EC, Smith R, 'Phasic phosphorylation of caldesmon and ERK 1/2 during contractions in human myometrium', PLoS ONE, 6 1-7 (2011) [C1]
DOI 10.1371/journal.pone.0021542
Citations Scopus - 16Web of Science - 13
Co-authors Roger Smith, Alexis Hure, Jonathan Paul
2011 Maiti K, Paul JW, Read MA, Chan EC, Riley SC, Nahar P, Smith R, 'G-1-activated membrane estrogen receptors mediate increased contractility of the human myometrium', Endocrinology, 152 2448-2455 (2011) [C1]
DOI 10.1210/en.2010-0979
Citations Scopus - 31Web of Science - 31
Co-authors Jonathan Paul, Roger Smith
2011 Chen Y, Allars MJ, Maiti K, Angeli GL, Abou-Seif C, Smith R, Nicholson RC, 'Factors affecting cytotrophoblast cell viability and differentiation: Evidence of a link between syncytialisation and apoptosis', International Journal of Biochemistry & Cell Biology, 43 821-828 (2011) [C1]
DOI 10.1016/j.biocel.2011.02.007
Citations Scopus - 9Web of Science - 7
Co-authors Roger Smith, Giavanna Angeli
2009 Kim D-K, Yang JS, Maiti K, Hwang J-I, Kim K, Seen D, et al., 'A Gonadotropin-Releasing Hormone-II Antagonist Induces Autophagy of Prostate Cancer Cells', CANCER RESEARCH, 69 923-931 (2009) [C1]
DOI 10.1158/0008-5472.CAN-08-2115
Citations Scopus - 37Web of Science - 35
2008 Wang C, Oh DY, Maiti K, Kwon HB, Cheon J, Hwang JI, Seong JY, 'Involvement of amino acids flanking Glu

The Glu/Asp7.32 residue in extracellular loop 3 of the mammalian type-I gonadotropin-releasing hormone receptor (GnRHR) interacts with Arg8 of GnRH-I, conferring preferential liga... [more]

The Glu/Asp7.32 residue in extracellular loop 3 of the mammalian type-I gonadotropin-releasing hormone receptor (GnRHR) interacts with Arg8 of GnRH-I, conferring preferential ligand selectivity for GnRH-I over GnRH-II. Previously, we demonstrated that the residues (Ser and Pro) flanking Glu/Asp7.32 also play a role in the differential agonist selectivity of mammalian and non-mammalian GnRHRs. In this study, we examined the differential antagonist selectivity of wild type and mutant GnRHRs in which the Ser and Pro residues were changed. Cetrorelix, a GnRH-I antagonist, and Trptorelix-2, a GnRH-II antagonist, exhibited high selectivity for mammalian type-I and non-mammalian GnRHRs, respectively. The inhibitory activities of the antagonists were dependent on agonist concentration and subtype. Rat GnRHR in which the Ser-Glu-Pro (SEP) motif was changed to Pro-Glu-Val (PEV) or Pro-Glu-Ser (PES) had increased sensitivity to Trptorelix-2 but decreased sensitivity to Cetrorelix. Mutant bullfrog GnRHR-1 with the SEP motif had the reverse antagonist selectivity, with redueed sensitivity to Trptorelix-2 but increased sensitivity to Cetrorelix. These findings indicate that the residues flanking Glu7.32 are important for antagonist as well as agonist selectivity. © KSMCB 2008.

2007 Eid AH, Maiti K, Mitra S, Chotani MA, Flavahan S, Bailey SR, et al., 'Estrogen increases smooth muscle expression of alpha(2C)-adrenoceptors and cold-induced constriction of cutaneous arteries', AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY, 293 H1955-H1961 (2007)
DOI 10.1152/ajpheart.00306.2007
Citations Scopus - 59Web of Science - 56
2005 Maiti K, Oh DY, Moon JS, Acharjee S, Li JH, Bai DG, et al., 'Differential effects of gonadotropin-releasing hormone (GnRH)-I and GnRH-II on prostate cancer cell signaling and death', JOURNAL OF CLINICAL ENDOCRINOLOGY & METABOLISM, 90 4287-4298 (2005)
DOI 10.1210/jc.2004-1894
Citations Web of Science - 20
2005 Li JH, Choe H, Wang AF, Maiti K, Wang CB, Salam A, et al., 'Extracellular loop 3 (EL3) and EL3-proximal transmembrane helix 7 of the mammalian type I and type II gonadotropin-releasing hormone (GnRH) receptors determine differential ligand selectivity to GnRH-I and GnRH-II', MOLECULAR PHARMACOLOGY, 67 1099-1110 (2005)
DOI 10.1124/mol.104.004887
Citations Scopus - 22Web of Science - 21
2004 Wang CB, Yun O, Maiti K, Oh DY, Kim KK, Chae CH, et al., 'Position of pro and Ser near Glu(7.32) in the extracellular loop 3 of mammalian and nonmammalian gonadotropin-releasing hormone (GnRH) receptors is a critical determinant for differential ligand selectivity for mammalian GnRH and chicken GnRH-II', MOLECULAR ENDOCRINOLOGY, 18 105-116 (2004)
DOI 10.1210/me.2003-0101
Citations Scopus - 32Web of Science - 27
2003 Maiti K, Li J, Wang AF, Acharjee S, Kim WP, Im WB, et al., 'GnRH-II analogs for selective activation and inhibition of non-mammalian and type-II mammalian GnRH receptors.', Molecules and cells, 16 173-179 (2003)
Citations Scopus - 20Web of Science - 21
2003 Seong JY, Wang L, Oh DY, Yun O, Maiti K, Li JH, et al., 'Ala/Thr(201) in extracellular loop 2 and Leu/Phe(290) in transmembrane domain 6 of type 1 frog gonadotropin-releasing hormone receptor confer differential ligand sensitivity and signal transduction', ENDOCRINOLOGY, 144 454-466 (2003)
DOI 10.1210/en.2002-220683
Citations Scopus - 40Web of Science - 36
2003 Wang AF, Li JH, Maiti K, Kim WP, Kang HM, Seong JY, Kwon HB, 'Preferential ligand selectivity of the monkey type-II gonadotropin-releasing hormone (GnRH) receptor for GnRH-2 and its analogs', MOLECULAR AND CELLULAR ENDOCRINOLOGY, 209 33-42 (2003)
DOI 10.1016/j.mce.2003.08.004
Citations Scopus - 17Web of Science - 14
2002 Acharjee S, Maiti K, Soh JM, Im WB, Seong JY, Kwon HB, 'Differential desensitization and internalization of three different bullfrog gonadotropin-releasing hormone receptors', Molecules and Cells, 14 101-107 (2002)

We previously demonstrated the presence of three distinct types of the gonadotropin-releasing hormone receptor (GnRHR) in a bullfrog (denoted bfGnRHR-1, bfGnRHR-2, and bfGnRHR-3).... [more]

We previously demonstrated the presence of three distinct types of the gonadotropin-releasing hormone receptor (GnRHR) in a bullfrog (denoted bfGnRHR-1, bfGnRHR-2, and bfGnRHR-3). The bfGnRHRs exhibited differential tissue distribution and ligand selectivity. In the present study, we demonstrated the desensitization and internalization kinetics of these receptors in both transiently-transfected HEK293 cells and retrovirus-mediated stable cells. The time-course accumulation of the inositol phosphate in response to GnRH revealed that bfGnRHR-1 and -2 were rapidly desensitized, whereas bfGnRHR-3 was slowly desensitized. A comparison of the internalization kinetics revealed the most rapid rate and highest extent of internalization of bfGnRHR-2 among the three receptors. Interestingly, the mechanisms that underlie the receptor internalization appear to differ from each other. Internalization of bfGnRHR-1 was dependent on both dynamin and ß-arrestin, whereas those of bfGnRHR-2 and -3 were dependent on dynamin, but not on ß-arrestin. These results, therefore, suggest that differential regulatory mechanisms for desensitization and internalization of the GnRHR are involved in diverse cellular and physiological responses to GnRH stimulation. ©KSMCB 2002.

Citations Scopus - 26
2001 Chattopadhyay D, Maiti K, Kundu AP, Chakraborty MS, Bhadra R, Mandal SC, Mandal AB, 'Antimicrobial activity of Alstonia macrophylla: a folklore of bay islands', JOURNAL OF ETHNOPHARMACOLOGY, 77 49-55 (2001)
DOI 10.1016/S0378-8741(01)00264-1
Citations Scopus - 72Web of Science - 53
Show 22 more journal articles

Conference (14 outputs)

Year Citation Altmetrics Link
2018 Hammer P, Attia J, Oldmeadow C, Holder C, Maiti K, Smith R, Morgan TK, 'Increased Erythropoiesis in Post-Dates Deliveries is Associated with Fetal Sex', REPRODUCTIVE SCIENCES, San Diego, CA (2018)
Co-authors John Attia, Roger Smith, Christopher Oldmeadow
2018 Sultana Z, Maiti K, Smith R, Hansbro P, Mononair P, 'Growth Factor Depletion in Placental Cells Increases Lipid Peroxidation and Reduces Mitochondrial Function and mTOR Activity via Aldehyde Oxidase Mediated Pathways', San Diego, CA (2018)
DOI 10.1177/1933719118759999
Co-authors Philip Hansbro, Zakia Sultana, Roger Smith
2017 Maiti K, Sultana Z, Aitken J, Smith R, 'Unexplained Antepartum Stillbirth Is Associated with Biochemical Evidence of Placental Aging', REPRODUCTIVE SCIENCES, Orlando, FL (2017)
DOI 10.1177/1933719117699773
Co-authors John Aitken, Zakia Sultana, Roger Smith
2016 Maiti K, Sultana Z, Aitken J, Smith R, 'Evidence That Stillbirth Is Linked to Placental Aging.', REPRODUCTIVE SCIENCES, Montreal, CANADA (2016)
DOI 10.1177/1933719116641257
Co-authors John Aitken, Zakia Sultana, Roger Smith
2015 Maiti K, Sultana Z, Aitken J, Smith R, 'THE HUMAN PLACENTA AT 41 WEEKS OF GESTATION SHOWS EVIDENCE OF AGING WITH SHORTENED TELOMERES, DNA OXIDATION AND CHANGES IN IGFR2, AUTOPHAGY AND MTOR', PLACENTA (2015) [E3]
DOI 10.1016/j.placenta.2015.07.334
Co-authors Zakia Sultana, John Aitken, Roger Smith
2015 Maiti K, Aitken J, Smith R, 'The Human Placenta at 41 Weeks of Gestation Shows Evidence of Aging With Shortened Telomeres, DNA Oxidation and Changes in IGFR2, Autophagy and the mTOR', REPRODUCTIVE SCIENCES (2015) [E3]
Co-authors Zakia Sultana, Roger Smith, John Aitken
2015 Sultana Z, Maiti K, Smith R, 'USE OF AN IN VITRO HUMAN PLACENTAL EXPLANT CULTURE MODEL TO INTERROGATE THE EFFECTS OF STARVATION ON PLACENTAL FUNCTION', PLACENTA (2015) [E3]
DOI 10.1016/j.placenta.2015.07.335
Co-authors Roger Smith, Zakia Sultana
2012 Maiti K, Gravina FS, Bowman M, Aitken RJ, Smith R, 'The ageing placenta and still birth', Abstracts. The Annual Scientific Meeting of the Endocrine Society of Australia and the Society for Reproductive Biology 2012, Gold Coast, QLD (2012) [E3]
Co-authors Roger Smith, Maria Bowman, John Aitken
2011 Paul JW, Maiti K, Read MA, Hure AJ, Smith JI, Chan EC, Smith R, 'Studying laboring myometrium misses phosphorylation changes associated with contraction', Reproductive Sciences, Miami Beach (2011) [E3]
Co-authors Jonathan Paul, Alexis Hure, Roger Smith
2010 Maiti K, Paul JW, Read MA, Chan EC, Smith R, 'Human labor Is associated with internalization and oligomerization of the membrane estrogen receptor, GPR30', Reproductive Sciences, Orlando, Florida (2010) [E3]
Co-authors Roger Smith, Jonathan Paul
2010 Maiti K, Paul JW, Chan EC, Smith R, 'GPR30, the novel membrane estrogen receptor, stimulates contractility of myometrium by increasing expression of h-caldesmon', The Endocrine Society of Australia Annual Scientific Meeting Meeting Proceedings and Abstract Book, Sydney (2010) [E3]
Co-authors Roger Smith, Jonathan Paul
2010 Paul JW, Maiti K, Read MA, Smith R, 'Caldesmon phosphorylation and phasic regulation of ERK 1/2 during contractions in human myometrium', The Endocrine Society of Australia Annual Scientific Meeting Proceedings and Abstract Book, Sydney (2010) [E3]
Co-authors Jonathan Paul, Roger Smith
2009 Maiti K, Paul JW, Read MA, Chan EC, Smith R, 'Demonstration that activation of the cell surface estrogen receptor GPR30 causes phosphorylation of HSP27 and MAPK p42/44 in term human myometrial tissue and explants', Reproductive Sciences, Glasgow, Scotland (2009) [E3]
Co-authors Jonathan Paul, Roger Smith
2008 Maiti K, Paul JW, Read MA, Chan EC, Smith R, 'Presence of the novel membrane estrogen receptor G-Protein coupled receptor 30 (GPR30) a membrane estrogen receptor in human pregnant myometrium and its biochemical characterization', 51st Annual Scientific Meeting of the Endocrine Society of Australia and Society of Reproductive Biology: Meeting Proceedings and Abstract Book, Melbourne, VIC (2008) [E3]
Co-authors Jonathan Paul, Roger Smith
Show 11 more conferences
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Grants and Funding

Summary

Number of grants 12
Total funding $2,244,834

Click on a grant title below to expand the full details for that specific grant.


20181 grants / $17,307

Measurement of cell free mRNA for corticotropin releasing hormone in maternal plasma to predict preterm labour and birth using a novel toehold switch RNA sensor$17,307

Funding body: John Hunter Hospital Charitable Trust

Funding body John Hunter Hospital Charitable Trust
Project Team Doctor Kaushik Maiti, Dr Wendy Carseldine
Scheme Research Grant
Role Lead
Funding Start 2018
Funding Finish 2018
GNo G1801083
Type Of Funding C2220 - Aust StateTerritoryLocal - Other
Category 2220
UON Y

20171 grants / $1,168,600

HMRI Research Fellowship in Stillbirth$1,168,600

Funding body: Hunter Medical Research Institute

Funding body Hunter Medical Research Institute
Project Team Laureate Professor Roger Smith, Doctor Kaushik Maiti
Scheme Project Grant
Role Investigator
Funding Start 2017
Funding Finish 2022
GNo G1701568
Type Of Funding C3120 - Aust Philanthropy
Category 3120
UON Y

20142 grants / $553,512

Is placental aging the key to understanding, predicting and preventing stillbirth$526,000

Funding body: Hunter Medical Research Institute

Funding body Hunter Medical Research Institute
Project Team Doctor Kaushik Maiti
Scheme Project Grant
Role Lead
Funding Start 2014
Funding Finish 2018
GNo G1400895
Type Of Funding C3120 - Aust Philanthropy
Category 3120
UON Y

Changes in mTOR and lysosomal signalling in post-dated placentas and its connection to Third-trimester Antepartum Still Birth$27,512

Funding body: John Hunter Hospital Charitable Trust

Funding body John Hunter Hospital Charitable Trust
Project Team Doctor Kaushik Maiti, Doctor Pravin Nahar
Scheme Research Grant
Role Lead
Funding Start 2014
Funding Finish 2014
GNo G1401147
Type Of Funding Other Public Sector - State
Category 2OPS
UON Y

20131 grants / $23,810

Potential of oxidised telomeres in maternal blood as a predictor of antepartum fetal death$23,810

Funding body: John Hunter Hospital Charitable Trust

Funding body John Hunter Hospital Charitable Trust
Project Team Doctor Kaushik Maiti, Doctor Pravin Nahar
Scheme Research Grant
Role Lead
Funding Start 2013
Funding Finish 2013
GNo G1300740
Type Of Funding Other Public Sector - State
Category 2OPS
UON Y

20122 grants / $44,454

The Ageing Placenta and Intrauterine Death$34,454

Funding body: John Hunter Hospital Charitable Trust

Funding body John Hunter Hospital Charitable Trust
Project Team Doctor Kaushik Maiti, Doctor Pravin Nahar
Scheme Research Grant
Role Lead
Funding Start 2012
Funding Finish 2012
GNo G1200641
Type Of Funding Other Public Sector - State
Category 2OPS
UON Y

Precellys Tissue Homogeniser$10,000

Funding body: NHMRC (National Health & Medical Research Council)

Funding body NHMRC (National Health & Medical Research Council)
Project Team Laureate Professor Roger Smith, Professor Eugenie Lumbers, Conjoint Professor Tamas Zakar, Professor Jon Hirst, Conjoint Professor Ian Wright, Doctor Gemma Madsen, Doctor Kaushik Maiti
Scheme Equipment Grant
Role Investigator
Funding Start 2012
Funding Finish 2012
GNo G1100979
Type Of Funding Other Public Sector - Commonwealth
Category 2OPC
UON Y

20111 grants / $69,157

Research microscope, confocal ready nikon eclipse 90i microscope$69,157

Funding body: NHMRC (National Health & Medical Research Council)

Funding body NHMRC (National Health & Medical Research Council)
Project Team Laureate Professor Roger Smith, Conjoint Professor Tamas Zakar, Professor Jon Hirst, Doctor Kaushik Maiti, Doctor Gemma Madsen, Laureate Professor Rodney Scott, Conjoint Professor Peter Wark, Laureate Professor Paul Foster, Professor Phil Hansbro, Conjoint Professor Ian Wright
Scheme Equipment Grant
Role Investigator
Funding Start 2011
Funding Finish 2011
GNo G1100024
Type Of Funding Other Public Sector - Commonwealth
Category 2OPC
UON Y

20101 grants / $324,900

Estrogen receptors and the onset of labour$324,900

Funding body: NHMRC (National Health & Medical Research Council)

Funding body NHMRC (National Health & Medical Research Council)
Project Team Laureate Professor Roger Smith, Doctor Kaushik Maiti
Scheme Project Grant
Role Investigator
Funding Start 2010
Funding Finish 2012
GNo G0190158
Type Of Funding Aust Competitive - Commonwealth
Category 1CS
UON Y

20092 grants / $33,494

The role of multiple forms of Estrogen and their Interaction with GPR30, a Novel Transmembrane Estrogen Receptor in Human Labour$18,286

Funding body: John Hunter Hospital Charitable Trust

Funding body John Hunter Hospital Charitable Trust
Project Team Doctor Kaushik Maiti, Doctor Pravin Nahar
Scheme Research Grant
Role Lead
Funding Start 2009
Funding Finish 2009
GNo G0900122
Type Of Funding Other Public Sector - State
Category 2OPS
UON Y

Apoptosis in pregnant human myometrium and its role in labour$15,208

Funding body: John Hunter Hospital Charitable Trust

Funding body John Hunter Hospital Charitable Trust
Project Team Doctor Kaushik Maiti, Doctor Jonathan Paul, Doctor Pravin Nahar
Scheme Research Grant
Role Lead
Funding Start 2009
Funding Finish 2009
GNo G0189933
Type Of Funding Other Public Sector - State
Category 2OPS
UON Y

20081 grants / $9,600

LED fluorescence illuminators and filter set (525nm + 575DF20) for LAS3000 image analysis system$9,600

Funding body: NHMRC (National Health & Medical Research Council)

Funding body NHMRC (National Health & Medical Research Council)
Project Team Laureate Professor Roger Smith, Professor Ian Symonds, Conjoint Associate Professor Andrew Bisits, Conjoint Professor Tamas Zakar, Doctor John Fitter, Doctor Eng-Cheng Chan, Conjoint Associate Professor Rick Nicholson, Doctor Giavanna Angeli, Doctor Kaushik Maiti, Doctor Jonathan Paul, Professor Jon Hirst, Doctor Hannah Palliser, Professor Eugenie Lumbers
Scheme Equipment Grant
Role Investigator
Funding Start 2008
Funding Finish 2008
GNo G0188543
Type Of Funding Other Public Sector - Commonwealth
Category 2OPC
UON Y
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Research Supervision

Number of supervisions

Completed1
Current2

Current Supervision

Commenced Level of Study Research Title Program Supervisor Type
2019 PhD Determining the Factors Regulating Placental Exosome Production and the Relationship Between Placental Pathology and Exosome Content PhD (Medical Biochemistry), Faculty of Health and Medicine, The University of Newcastle Co-Supervisor
2017 PhD Development of AOX1 Inhibitors PhD (Reproductive Medicine), Faculty of Health and Medicine, The University of Newcastle Co-Supervisor

Past Supervision

Year Level of Study Research Title Program Supervisor Type
2018 PhD Increased Oxidative Damage and Premature Placental Aging Contribute to the Aetiology of Stillbirth PhD (Reproductive Medicine), Faculty of Health and Medicine, The University of Newcastle Co-Supervisor
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Dr Kaushik Maiti

Position

Research Officer
Mother and Babies Research Centre
School of Medicine and Public Health
Faculty of Health and Medicine

Contact Details

Email kaushik.maiti@newcastle.edu.au
Phone (02) 4042 0347
Mobile 0402197903

Office

Room HMRI, 3502
Location Hunter Medical Research Institute, New lambton Heights, Newcastle Level 3 East

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